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Watering Collecting Duct Cysts: Are Aquaporin2+ Progenitors the Answer?

作者信息

Ostrosky-Frid Mauricio, Patel Vishal

机构信息

Division of Nephrology, Department of Internal Medicine, UT Southwestern Medical Center, Dallas, Texas.

出版信息

J Am Soc Nephrol. 2024 Apr 1;35(4):387-388. doi: 10.1681/ASN.0000000000000333. Epub 2024 Mar 13.

DOI:10.1681/ASN.0000000000000333
PMID:38557785
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11000719/
Abstract
摘要

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Watering Collecting Duct Cysts: Are Aquaporin2+ Progenitors the Answer?集合管囊肿：水通道蛋白2+祖细胞是答案吗？
J Am Soc Nephrol. 2024 Apr 1;35(4):387-388. doi: 10.1681/ASN.0000000000000333. Epub 2024 Mar 13.
2
Histogenesis of the renal cysts in adult (autosomal dominant) polycystic kidney disease: a histochemical study.成人（常染色体显性）多囊肾病中肾囊肿的组织发生：一项组织化学研究。
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The SGP-2 gene is developmentally regulated in the mouse kidney and abnormally expressed in collecting duct cysts in polycystic kidney disease.SGP-2基因在小鼠肾脏中受发育调控，在多囊肾病的集合管囊肿中异常表达。
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Am J Pathol. 2006 Dec;169(6):1925-38. doi: 10.2353/ajpath.2006.060245.

本文引用的文献

1
Pkd2 Deficiency in Embryonic Aqp2 + Progenitor Cells Is Sufficient to Cause Severe Polycystic Kidney Disease.PKD2 缺失在胚胎 AQP2+祖细胞中足以导致严重的多囊肾病。
J Am Soc Nephrol. 2024 Apr 1;35(4):398-409. doi: 10.1681/ASN.0000000000000309. Epub 2024 Jan 23.
2
Aqp2 Progenitor Cells Maintain and Repair Distal Renal Segments.水通道蛋白 2 祖细胞维持和修复远端肾单位。
J Am Soc Nephrol. 2022 Jul;33(7):1357-1376. doi: 10.1681/ASN.2021081105. Epub 2022 Mar 22.
3
Loss of oriented cell division does not initiate cyst formation.定向细胞分裂的丧失并不会引发囊肿的形成。
J Am Soc Nephrol. 2010 Feb;21(2):295-302. doi: 10.1681/ASN.2009060603. Epub 2009 Dec 3.
4
Advances in the pathogenesis and treatment of polycystic kidney disease.多囊肾病的发病机制与治疗进展
Curr Opin Nephrol Hypertens. 2009 Mar;18(2):99-106. doi: 10.1097/MNH.0b013e3283262ab0.
5
Inactivation of Pkd1 in principal cells causes a more severe cystic kidney disease than in intercalated cells.在主细胞中Pkd1的失活比在闰细胞中导致更严重的多囊肾病。
Kidney Int. 2009 Mar;75(6):626-33. doi: 10.1038/ki.2008.659. Epub 2009 Jan 14.
6
Acute kidney injury and aberrant planar cell polarity induce cyst formation in mice lacking renal cilia.急性肾损伤和异常的平面细胞极性在缺乏肾纤毛的小鼠中诱导囊肿形成。
Hum Mol Genet. 2008 Jun 1;17(11):1578-90. doi: 10.1093/hmg/ddn045. Epub 2008 Feb 9.
7
A critical developmental switch defines the kinetics of kidney cyst formation after loss of Pkd1.一个关键的发育开关决定了Pkd1缺失后肾囊肿形成的动力学。
Nat Med. 2007 Dec;13(12):1490-5. doi: 10.1038/nm1675. Epub 2007 Oct 28.