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本文引用的文献

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Mutations, inflammation and phenotype of myeloproliferative neoplasms.
Front Oncol. 2023 May 22;13:1196817. doi: 10.3389/fonc.2023.1196817. eCollection 2023.
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NF-κB in monocytes and macrophages - an inflammatory master regulator in multitalented immune cells.
Front Immunol. 2023 Feb 23;14:1134661. doi: 10.3389/fimmu.2023.1134661. eCollection 2023.
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JAK/STAT pathway: Extracellular signals, diseases, immunity, and therapeutic regimens.
Front Bioeng Biotechnol. 2023 Feb 23;11:1110765. doi: 10.3389/fbioe.2023.1110765. eCollection 2023.
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CXCL8/CXCR2 signaling mediates bone marrow fibrosis and is a therapeutic target in myelofibrosis.
Blood. 2023 May 18;141(20):2508-2519. doi: 10.1182/blood.2022015418.
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The Role of JAK/STAT Pathway in Fibrotic Diseases: Molecular and Cellular Mechanisms.
Biomolecules. 2023 Jan 6;13(1):119. doi: 10.3390/biom13010119.
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IL-13/IL-4 signaling contributes to fibrotic progression of the myeloproliferative neoplasms.
Blood. 2022 Dec 29;140(26):2805-2817. doi: 10.1182/blood.2022017326.
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GLI1 activates pro-fibrotic pathways in myelofibrosis fibrocytes.
Cell Death Dis. 2022 May 20;13(5):481. doi: 10.1038/s41419-022-04932-4.
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Defining disease modification in myelofibrosis in the era of targeted therapy.
Cancer. 2022 Jul 1;128(13):2420-2432. doi: 10.1002/cncr.34205. Epub 2022 May 2.

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