Sancilio Christopher, Mastroianni Michael, Mousad Albert, Jesus Nicholas De, McCormick Frank
Department of Biology, Florida Atlantic University, Boca Raton, Florida.
Columbia University Irving Medical Center/New York Presbyterian Hospital, New York, United States.
J Orthop Case Rep. 2024 Apr;14(4):67-72. doi: 10.13107/jocr.2024.v14.i04.4362.
A patient presented for recalcitrant right hip pain secondary to femoroacetabular impingement (FAI) after blunt motor vehicle trauma and following the development of a 12 cm heterotopic ossification (HO). FAI is an increasingly recognized diagnosis where the hip joint is exposed to repeated femoral microtrauma from high-level physical activity or trauma, often causing labral ossification, and perhaps underlying a similar biological mechanism to HO.
In this case report, we have an otherwise healthy 49-year-old male who was involved in a high-speed motor vehicle collision who was diagnosed with right hip FAI secondary to HO (Brooker's Class IV) and indicated for surgical excision of the HO anterior to the right proximal femur. The care team and patient initially trialed non-operative conservative treatment with non-steroidal anti-inflammatories drugs (NSAIDs) and hypothesized therapeutic success using a non-surgical approach. Surgical resection was pursued with the patient after a failure of conservative measures. The patient reported a zero out of ten on a ten-point numerical rating scale for pain, he also stated improved quality of life, satisfaction with the procedure, and subsequent rehabilitation at 1-month post-operative follow-up.
HO with near complete ankylosis of the hip joint may be causative of FAI when untreated. Although this case demonstrates a rarely studied traumatic etiology of impingement secondary to HO, initial standard conservative anti-inflammatory treatment can still be pursued. By analyzing the periarticular impact of HO secondary to non-surgical trauma, we can utilize and make inferential correlations from the literature, studying HO and impingement in the setting of prior hip surgery to guide treatment and prognosis in those presenting with FAI symptoms secondary to blunt force trauma.
一名患者因钝性机动车创伤后继发股骨髋臼撞击症(FAI)以及出现12厘米的异位骨化(HO)而出现顽固性右髋疼痛。FAI是一种越来越被认可的诊断,髋关节因高水平体育活动或创伤而反复遭受股骨微创伤,常导致盂唇骨化,可能与HO存在相似的生物学机制。
在本病例报告中,我们有一名49岁的健康男性,他曾遭遇高速机动车碰撞,被诊断为继发于HO(布鲁克四级)的右髋FAI,并被建议对右股骨近端前方的HO进行手术切除。护理团队和患者最初尝试使用非甾体抗炎药(NSAIDs)进行非手术保守治疗,并假设采用非手术方法会取得治疗成功。保守措施失败后,患者接受了手术切除。患者在术后1个月的随访中报告,其疼痛在10分制数字评分量表上为零分,他还表示生活质量有所改善,对手术和后续康复感到满意。
髋关节几乎完全强直的HO如果不治疗可能是FAI的病因。尽管本病例展示了一种很少被研究的继发于HO的创伤性撞击病因,但仍可首先采用标准的保守抗炎治疗。通过分析非手术创伤继发的HO对关节周围的影响,我们可以利用文献并进行推断性关联,研究既往髋关节手术背景下的HO和撞击,以指导对钝性暴力创伤继发FAI症状患者的治疗和预后。