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Gravi-D peptide disrupts HDAC11 association with an AKAP to stimulate adipocyte thermogenic signaling.

作者信息

Robinson Emma L, Tharp Charles A, Bagchi Rushita A, McKinsey Timothy A

机构信息

Department of Medicine, Division of Cardiology and.

Consortium for Fibrosis Research & Translation, University of Colorado Anschutz Medical Campus, Aurora, Colorado, USA.

出版信息

J Clin Invest. 2024 May 1;134(9):e177726. doi: 10.1172/JCI177726.

DOI:10.1172/JCI177726
PMID:38690735
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11060728/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/abcd/11060728/a20244ccaa8a/jci-134-177726-g134.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/abcd/11060728/a20244ccaa8a/jci-134-177726-g134.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/abcd/11060728/a20244ccaa8a/jci-134-177726-g134.jpg

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2
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本文引用的文献

1
HDAC11 inhibition triggers bimodal thermogenic pathways to circumvent adipocyte catecholamine resistance.组蛋白去乙酰化酶 11 抑制触发双模态产热途径以规避脂肪细胞儿茶酚胺抵抗。
J Clin Invest. 2023 Oct 2;133(19):e168192. doi: 10.1172/JCI168192.
2
Reversible lysine fatty acylation of an anchoring protein mediates adipocyte adrenergic signaling.锚定蛋白赖氨酸可逆脂肪酸酰化介导脂肪细胞肾上腺素能信号转导。
Proc Natl Acad Sci U S A. 2022 Feb 15;119(7). doi: 10.1073/pnas.2119678119.
3
β3-Adrenergic receptor downregulation leads to adipocyte catecholamine resistance in obesity.
β3-肾上腺素能受体下调导致肥胖症中脂肪细胞儿茶酚胺抵抗。
J Clin Invest. 2022 Jan 18;132(2). doi: 10.1172/JCI153357.
4
HDAC11 regulates type I interferon signaling through defatty-acylation of SHMT2.HDAC11 通过去脂酰化 SHMT2 调节 I 型干扰素信号。
Proc Natl Acad Sci U S A. 2019 Mar 19;116(12):5487-5492. doi: 10.1073/pnas.1815365116. Epub 2019 Feb 28.
5
Identification of novel phosphorylation sites in hormone-sensitive lipase that are phosphorylated in response to isoproterenol and govern activation properties in vitro.鉴定激素敏感脂肪酶中新型磷酸化位点,这些位点在异丙肾上腺素作用下发生磷酸化并在体外调控激活特性。
J Biol Chem. 1998 Jan 2;273(1):215-21. doi: 10.1074/jbc.273.1.215.