Endothelial-Mesenchymal Transition Switch and Diabetic Kidney Disease.
作者信息
Doke Tomohito, Bjornstad Petter
机构信息
Department of Nephrology, Nagoya University Graduate School of Medicine, Nagoya, Japan.
Department of Pediatrics, Section of Endocrinology, University of Colorado School of Medicine, Aurora, Colorado.
出版信息
J Am Soc Nephrol. 2024 Jun 1;35(6):670-672. doi: 10.1681/ASN.0000000000000364. Epub 2024 May 2.
Abstract
摘要
相似文献
1
Endothelial-Mesenchymal Transition Switch and Diabetic Kidney Disease.内皮-间充质转化开关与糖尿病肾病
J Am Soc Nephrol. 2024 Jun 1;35(6):670-672. doi: 10.1681/ASN.0000000000000364. Epub 2024 May 2.
2
FSP1-specific SMAD2 knockout in renal tubular, endothelial, and interstitial cells reduces fibrosis and epithelial-to-mesenchymal transition in murine STZ-induced diabetic nephropathy.FSP1 特异性 SMAD2 敲除在肾小管、内皮和间质细胞中可减少 STZ 诱导的糖尿病肾病小鼠的纤维化和上皮间质转化。
Cell Tissue Res. 2018 Apr;372(1):115-133. doi: 10.1007/s00441-017-2754-1. Epub 2017 Dec 6.
3
Empagliflozin attenuating renal interstitial fibrosis in diabetic kidney disease by inhibiting lymphangiogenesis and lymphatic endothelial-to-mesenchymal transition via the VEGF-C/VEGFR3 pathway.恩格列净通过抑制血管内皮生长因子 C/血管内皮生长因子受体 3 通路抑制淋巴管生成和淋巴管内皮细胞向间充质转化来减轻糖尿病肾病中的肾间质纤维化。
Biomed Pharmacother. 2024 Nov;180:117589. doi: 10.1016/j.biopha.2024.117589. Epub 2024 Oct 16.
4
SirT7-mediated transcription of fascin in hyperglycemic glomerular endothelial cells contributes to EndMT in diabetic nephropathy.SirT7介导的高血糖肾小球内皮细胞中丝状肌动蛋白的转录促成糖尿病肾病中的内皮-间充质转化。
Acta Biochim Biophys Sin (Shanghai). 2024 Apr 25;56(4):586-596. doi: 10.3724/abbs.2024002.
5
Sirt7/HIC1 complex participates in hyperglycaemia-mediated EndMT via modulation of SDC1 expression in diabetic kidney disease and metabolic memory.Sirt7/HIC1 复合物通过调节糖尿病肾病和代谢记忆中 SDC1 的表达参与高血糖介导的 EndMT。
J Cell Mol Med. 2024 May;28(9):e18336. doi: 10.1111/jcmm.18336.
6
Loss of endothelial glucocorticoid receptor accelerates diabetic nephropathy.内皮糖皮质激素受体缺失加速糖尿病肾病。
Nat Commun. 2021 Apr 22;12(1):2368. doi: 10.1038/s41467-021-22617-y.
7
Research progress of endothelial-mesenchymal transition in diabetic kidney disease.糖尿病肾病中内皮-间充质转化的研究进展。
J Cell Mol Med. 2022 Jun;26(12):3313-3322. doi: 10.1111/jcmm.17356. Epub 2022 May 13.
8
Review: Endothelial-myofibroblast transition, a new player in diabetic renal fibrosis.综述：内皮-肌成纤维细胞转分化，糖尿病肾病纤维化的新角色。
Nephrology (Carlton). 2010 Aug;15(5):507-12. doi: 10.1111/j.1440-1797.2010.01319.x.
9
Melatonin Attenuates Endothelial-to-Mesenchymal Transition of Glomerular Endothelial Cells via Regulating miR-497/ROCK in Diabetic Nephropathy.褪黑素通过调控糖尿病肾病中肾小球内皮细胞的miR-497/ROCK减轻内皮-间充质转化
Kidney Blood Press Res. 2018;43(5):1425-1436. doi: 10.1159/000493380. Epub 2018 Sep 13.
10
Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy.糖尿病肾小球病中足细胞病和蛋白尿的发病机制。
Curr Diabetes Rev. 2008 Feb;4(1):39-45. doi: 10.2174/157339908783502370.
引用本文的文献
1
Cellular cross-talk drives mesenchymal transdifferentiation in diabetic kidney disease.细胞间相互作用驱动糖尿病肾病中的间充质转分化。
Front Med (Lausanne). 2025 Jan 7;11:1499473. doi: 10.3389/fmed.2024.1499473. eCollection 2024.
Zotero 插件