Response by Gao et al to Letter Regarding Article, "Reduced Mitochondrial Protein Translation Promotes Cardiomyocyte Proliferation and Heart Regeneration".
作者信息
Gao Feng, Mably John D, Wang Da-Zhi, Chen Jinghai
机构信息
Department of Cardiology, State Key Laboratory of Transvascular Implantation Devices, Second Affiliated Hospital (F.G., J.C.), Zhejiang University School of Medicine, Hangzhou, China.
Institute of Translational Medicine (F.G., J.C.), Zhejiang University School of Medicine, Hangzhou, China.
出版信息
Circulation. 2024 May 21;149(21):e1195-e1196. doi: 10.1161/CIRCULATIONAHA.124.069377. Epub 2024 May 20.
Abstract
摘要
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本文引用的文献
1
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Circulation. 2023 Dec 5;148(23):1887-1906. doi: 10.1161/CIRCULATIONAHA.122.061192. Epub 2023 Oct 31.
2
A defect in mitochondrial protein translation influences mitonuclear communication in the heart.线粒体蛋白翻译缺陷影响心脏的线粒体-核通讯。
Nat Commun. 2023 Mar 22;14(1):1595. doi: 10.1038/s41467-023-37291-5.
3
Tetracycline-induced mitohormesis mediates disease tolerance against influenza.四环素有诱导mitohormesis 的作用，介导了对流感的疾病耐受。
J Clin Invest. 2022 Sep 1;132(17). doi: 10.1172/JCI151540.
4
The Antibiotic Doxycycline Impairs Cardiac Mitochondrial and Contractile Function.多西环素抗生素损害心肌线粒体和收缩功能。
Int J Mol Sci. 2021 Apr 15;22(8):4100. doi: 10.3390/ijms22084100.
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Mitonuclear protein imbalance as a conserved longevity mechanism.线粒体-核蛋白失衡作为一种保守的长寿机制。
Nature. 2013 May 23;497(7450):451-7. doi: 10.1038/nature12188.
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