Bouchez B, Arnott G, Delandsheer E, Blond S, Guieu J D
Acta Neurol Belg. 1985 Nov-Dec;85(5):269-76.
A case of femoral palsy associated with chronic pain sensible to Carbamazepine, secondary to iliacus haematoma during anticoagulant therapy is reported. Late femoral nerve decompression followed by transcutaneous neurostimulation permit the normalization of the sensory nerve conduction and a complete clinical recovery. The localizations of hemorrhage and nervous compression are discussed. The necessity of a nerve decompression when features of nervous conduction persist is emphasized. Clinical symptomatology, Carbamazepine and transcutaneous neurostimulation efficacity, electrophysiological features are correlated with a central deafferentation state caused by an incomplete and heterogeneous lesion of the sensory nerve conduction pathways.
本文报告1例股神经麻痹病例,该病例伴有对卡马西平敏感的慢性疼痛,继发于抗凝治疗期间的髂腰肌血肿。晚期股神经减压并辅以经皮神经刺激,使感觉神经传导恢复正常,临床完全康复。文中讨论了出血和神经受压的部位。强调当神经传导特征持续存在时进行神经减压的必要性。临床症状、卡马西平和经皮神经刺激的疗效、电生理特征与感觉神经传导通路不完全和异质性病变引起的中枢性传入阻滞状态相关。