Zhu S X, Lu S B, Yao J X, Liu Z S, Sun Y W, Wang X L, Li Z Y, Fu Z G, Liu Z X
General Hospital of PLA, Beijing, China.
Ann Plast Surg. 1985 Dec;15(6):460-4. doi: 10.1097/00000637-198512000-00002.
A new concept of the pathogenesis of causalgia is suggested in accordance with experimental and clinical work. Endoneural hypertension secondary to nerve trunk disruption is incriminated and intrafascicular decompression is used as the key therapeutic measure. In 14 patients with causalgia thus treated, all except 1 were cured. No symptomatic recurrence was noted in a 4- to 6-year follow-up study.
根据实验和临床研究成果，提出了一种关于灼性神经痛发病机制的新观点。认为继发于神经干断裂后的神经内高压是病因所在，而束内减压术则被用作关键治疗措施。在接受该治疗的14例灼性神经痛患者中，除1例之外全部治愈。在为期4至6年的随访研究中未发现症状复发情况。
