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利用整合微电极阵列的图案化诱导多能干细胞衍生的皮质神经元,对四种治疗阿尔茨海默病的药物进行功能性人类阿尔茨海默病模型的验证。

Validation of a functional human AD model with four AD therapeutics utilizing patterned iPSC-derived cortical neurons integrated with microelectrode arrays.

作者信息

Caneus Julbert, Autar Kaveena, Akanda Nesar, Grillo Marcella, Long Chris, Jackson Max, Lindquist Sarah, Guo Xiufang, Morgan Dave, Hickman James J

机构信息

University of Central Florida.

Hesperos Inc.

出版信息

Res Sq. 2024 May 20:rs.3.rs-4313679. doi: 10.21203/rs.3.rs-4313679/v1.

DOI:10.21203/rs.3.rs-4313679/v1
PMID:38826367
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11142300/
Abstract

Preclinical methods are needed for screening potential Alzheimer's disease (AD) therapeutics that recapitulate phenotypes found in the Mild Cognitive Impairment (MCI) stage or even before this stage of the disease. This would require a phenotypic system that reproduces cognitive deficits without significant neuronal cell death to mimic the clinical manifestations of AD during these stages. A potential functional parameter to be monitored is long-term potentiation (LTP), which is a correlate of learning and memory, that would be one of the first functions effected by AD onset. Mature human iPSC-derived cortical neurons and primary astrocytes were co-cultured on microelectrode arrays (MEA) where surface chemistry was utilized to create circuit patterns connecting two adjacent electrodes to model LTP function. LTP maintenance was significantly reduced in the presence of Amyloid-Beta 42 (Aβ42) oligomers compared to the controls, however, co-treatment with AD therapeutics (Donepezil, Memantine, Rolipram and Saracatinib) corrected Aβ42 induced LTP impairment. The results presented here illustrate the significance of the system as a validated platform that can be utilized to model and study MCI AD pathology, and potentially for the pre-MCI phase before the occurrence of significant cell death. It also has the potential to become an ideal platform for high content therapeutic screening for other neurodegenerative diseases.

摘要

需要临床前方法来筛选潜在的阿尔茨海默病(AD)治疗药物,这些药物要能重现轻度认知障碍(MCI)阶段甚至疾病此阶段之前所发现的表型。这将需要一个表型系统,该系统能再现认知缺陷且无明显神经元细胞死亡,以模拟AD在这些阶段的临床表现。一个有待监测的潜在功能参数是长时程增强(LTP),它与学习和记忆相关,是AD发病首先影响的功能之一。将成熟的人诱导多能干细胞衍生的皮质神经元和原代星形胶质细胞在微电极阵列(MEA)上共培养,利用表面化学方法创建连接两个相邻电极的电路模式以模拟LTP功能。与对照组相比,在存在β淀粉样蛋白42(Aβ42)寡聚体的情况下,LTP维持显著降低,然而,与AD治疗药物(多奈哌齐、美金刚、罗匹尼罗和萨拉卡替尼)联合处理可纠正Aβ42诱导的LTP损伤。此处呈现的结果说明了该系统作为一个经过验证的平台的重要性,该平台可用于模拟和研究MCI AD病理学,甚至可能用于重大细胞死亡发生之前的MCI前期。它还有潜力成为用于其他神经退行性疾病高内涵治疗筛选的理想平台。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/f883928d1e3b/nihpp-rs4313679v1-f0006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/26e2be8db845/nihpp-rs4313679v1-f0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/970ab17b3c54/nihpp-rs4313679v1-f0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/b9bd3552d9f9/nihpp-rs4313679v1-f0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/afdcb9118596/nihpp-rs4313679v1-f0004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/59b9882c87b1/nihpp-rs4313679v1-f0005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/f883928d1e3b/nihpp-rs4313679v1-f0006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/26e2be8db845/nihpp-rs4313679v1-f0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/970ab17b3c54/nihpp-rs4313679v1-f0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/b9bd3552d9f9/nihpp-rs4313679v1-f0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/afdcb9118596/nihpp-rs4313679v1-f0004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/59b9882c87b1/nihpp-rs4313679v1-f0005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/4bda/11142300/f883928d1e3b/nihpp-rs4313679v1-f0006.jpg

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