关注声明：乳腺肿瘤起始细胞中miR-128表达降低通过Bmi-1和ABCC5诱导化疗耐药 - Suppr

Expression of Concern: Reduced miR-128 in Breast Tumor-Initiating Cells Induces Chemotherapeutic Resistance via Bmi-1 and ABCC5.

作者信息

Zhu Yinghua, Yu Fengyan, Jiao Yu, Feng Juan, Tang Wei, Yao Herui, Gong Chang, Chen Jianing, Su Fengxi, Zhang Yan, Song Erwei

出版信息

Clin Cancer Res. 2024 Jul 1;30(13):2847. doi: 10.1158/1078-0432.CCR-24-1528.

DOI:10.1158/1078-0432.CCR-24-1528

PMID:38946292

Abstract

摘要

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Expression of Concern: Reduced miR-128 in Breast Tumor-Initiating Cells Induces Chemotherapeutic Resistance via Bmi-1 and ABCC5.关注声明：乳腺肿瘤起始细胞中miR-128表达降低通过Bmi-1和ABCC5诱导化疗耐药

Clin Cancer Res. 2024 Jul 1;30(13):2847. doi: 10.1158/1078-0432.CCR-24-1528.

Reduced miR-128 in breast tumor-initiating cells induces chemotherapeutic resistance via Bmi-1 and ABCC5.乳腺肿瘤起始细胞中 miR-128 的减少通过 Bmi-1 和 ABCC5 诱导化疗耐药。

Clin Cancer Res. 2011 Nov 15;17(22):7105-15. doi: 10.1158/1078-0432.CCR-11-0071. Epub 2011 Sep 27.

Editor's Note: Reduced miR-128 in Breast Tumor-Initiating Cells Induces Chemotherapeutic Resistance via Bmi-1 and ABCC5.编者按：乳腺肿瘤起始细胞中miR-128的减少通过Bmi-1和ABCC5诱导化疗耐药。

Clin Cancer Res. 2023 Jul 14;29(14):2738. doi: 10.1158/1078-0432.CCR-23-1698.

Enhanced SLC34A2 in breast cancer stem cell-like cells induces chemotherapeutic resistance to doxorubicin via SLC34A2-Bmi1-ABCC5 signaling.乳腺癌干细胞样细胞中增强的溶质载体家族34成员2（SLC34A2）通过SLC34A2-多梳蛋白抑制因子1（Bmi1）-ATP结合盒转运体C5（ABCC5）信号通路诱导对阿霉素的化疗耐药。

Tumour Biol. 2016 Apr;37(4):5049-62. doi: 10.1007/s13277-015-4226-0. Epub 2015 Nov 6.

Circ_0007031 enhances tumor progression and promotes 5-fluorouracil resistance in colorectal cancer through regulating miR-133b/ABCC5 axis.环状 RNA 0007031 通过调控 miR-133b/ABCC5 轴促进结直肠癌细胞的进展并增强其对 5-氟尿嘧啶的耐药性。

Cancer Biomark. 2020;29(4):531-542. doi: 10.3233/CBM-200023.

Human drug efflux transporter ABCC5 confers acquired resistance to pemetrexed in breast cancer.人类药物外排转运体ABCC5赋予乳腺癌对培美曲塞的获得性耐药性。

Cancer Cell Int. 2021 Feb 25;21(1):136. doi: 10.1186/s12935-021-01842-x.

Loss of miR-516a-3p mediates upregulation of ABCC5 in prostate cancer and drives its progression.miR-516a-3p缺失介导前列腺癌中ABCC5的上调并推动其进展。

Onco Targets Ther. 2018 Jul 6;11:3853-3867. doi: 10.2147/OTT.S167463. eCollection 2018.

ABCC5 supports osteoclast formation and promotes breast cancer metastasis to bone.ABCC5支持破骨细胞形成并促进乳腺癌向骨转移。

Breast Cancer Res. 2012 Nov 22;14(6):R149. doi: 10.1186/bcr3361.

Deregulation of miR-128 in ovarian cancer promotes cisplatin resistance.卵巢癌中miR-128的失调会促进顺铂耐药。

Int J Gynecol Cancer. 2014 Oct;24(8):1381-8. doi: 10.1097/IGC.0000000000000252.

Non-drug efflux function of ABCC5 promotes enzalutamide resistance in castration-resistant prostate cancer via upregulation of P65/AR-V7.ABCC5的非药物外排功能通过上调P65/AR-V7促进去势抵抗性前列腺癌中的恩杂鲁胺耐药。

Cell Death Discov. 2022 May 3;8(1):241. doi: 10.1038/s41420-022-00951-4.

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miR-196b strictly regulates and reliably predicts the response to cetuximab in colorectal cancer.微小RNA-196b严格调控并可靠预测结直肠癌对西妥昔单抗的反应。

Cell Mol Biol Lett. 2025 May 25;30(1):64. doi: 10.1186/s11658-025-00740-8.

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Expression of Concern: Reduced miR-128 in Breast Tumor-Initiating Cells Induces Chemotherapeutic Resistance via Bmi-1 and ABCC5.

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