Kraus Matthew J, Nguyen Joseph
Penn State College of Medicine, Hershey, PA, USA.
Case Rep Neurol. 2024 May 22;16(1):142-147. doi: 10.1159/000539405. eCollection 2024 Jan-Dec.
Spinal cord infarction is a rare but serious neurologic complication of spinal anesthesia. Direct vessel injury, intra-arterial anesthetic injection, and anesthetic-induced local hypotension are potential mechanisms of infarction during this procedure. The proximity of the artery of Adamkiewicz to the spinal levels used for spinal anesthesia may also play a role. This case of unilateral anterior spinal artery syndrome highlights the potential for an atypical pattern of injury and deficits due to the complexity of the spinal cord's anterior circulation.
We present a 38-year-old female patient who presented with left lower extremity weakness, loss of temperature sensation, and urinary retention following spinal anesthesia for cesarian section. Magnetic resonance imaging of the spine demonstrated T2 hyperintensities in the left central spinal cord from T8 to the conus medullaris. A diagnosis of spinal cord infarction was made after lumbar puncture testing showed no evidence of inflammatory myelitis. The patient was treated with steroids empirically until lumbar puncture testing showed no inflammation. The patient was discharged on daily aspirin with persistent left lower extremity weakness and loss of temperature sensation. A plan for outpatient physical therapy was made for rehabilitation.
Awareness of the potential for spinal cord infarction secondary to spinal anesthesia must increase among anesthesiologists, obstetricians, and neurologists. The risk of systemic hypotension during and after spinal anesthesia is important to recognize for both primary and secondary prevention of this complication. The hyperacute onset of myelopathic symptoms should point neurologists to investigate an ischemic etiology in the proper clinical context.
脊髓梗死是脊髓麻醉罕见但严重的神经并发症。直接血管损伤、动脉内注射麻醉剂以及麻醉引起的局部低血压是该过程中梗死的潜在机制。Adamkiewicz动脉与用于脊髓麻醉的脊髓节段的接近度也可能起作用。该例单侧脊髓前动脉综合征突出了由于脊髓前循环的复杂性而导致非典型损伤模式和功能缺损的可能性。
我们报告一名38岁女性患者,在剖宫产脊髓麻醉后出现左下肢无力、温度觉丧失和尿潴留。脊柱磁共振成像显示从T8至脊髓圆锥的左侧脊髓中央T2高信号。腰椎穿刺检查未发现炎性脊髓炎证据后,诊断为脊髓梗死。患者经验性使用类固醇治疗,直至腰椎穿刺检查显示无炎症。患者出院时服用每日阿司匹林,仍有持续的左下肢无力和温度觉丧失。制定了门诊物理治疗计划以进行康复。
麻醉医生、产科医生和神经科医生必须提高对脊髓麻醉继发脊髓梗死可能性的认识。认识脊髓麻醉期间和之后全身低血压的风险对于该并发症的一级和二级预防都很重要。脊髓病症状的超急性发作应促使神经科医生在适当的临床背景下调查缺血性病因。