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Response to letter about "Potential mechanisms of acyl-ghrelin in heart failure: insights into cholinergic modulation".

作者信息

Erhardsson Mikael, Ljung Faxén Ulrika, Lund Lars H

机构信息

Department of Medicine, Unit of Cardiology, Karolinska Institutet, Stockholm, Sweden.

Laboratory for Clinical Chemistry and Transfusion Medicine, Gävle Hospital, Gävle, Sweden.

出版信息

ESC Heart Fail. 2024 Oct;11(5):3453-3454. doi: 10.1002/ehf2.14992. Epub 2024 Jul 31.


DOI:10.1002/ehf2.14992
PMID:39082129
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11424354/
Abstract
摘要

相似文献

[1]
Response to letter about "Potential mechanisms of acyl-ghrelin in heart failure: insights into cholinergic modulation".

ESC Heart Fail. 2024-10

[2]
Potential mechanisms of acyl-ghrelin in heart failure: Insights into cholinergic modulation.

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本文引用的文献

[1]
Potential mechanisms of acyl-ghrelin in heart failure: Insights into cholinergic modulation.

ESC Heart Fail. 2024-10

[2]
Acyl ghrelin increases cardiac output while preserving right ventricular-pulmonary arterial coupling in heart failure.

ESC Heart Fail. 2024-2

[3]
Acyl-ghrelin therapy for heart failure: already a novel inotrope or even more?

Eur Heart J. 2023-6-9

[4]
Acyl ghrelin improves cardiac function in heart failure and increases fractional shortening in cardiomyocytes without calcium mobilization.

Eur Heart J. 2023-6-9

[5]
Vagus Nerve Stimulation Provides Multiyear Improvements in Autonomic Function and Cardiac Electrical Stability in the ANTHEM-HF Study.

J Card Fail. 2021-2

[6]
Effect of ghrelin on autonomic activity in healthy volunteers.

Peptides. 2014-12

[7]
Cholinergic activity as a new target in diseases of the heart.

Mol Med. 2015-1-26

[8]
Exogenous ghrelin improves blood flow distribution in pulmonary hypertension-assessed using synchrotron radiation microangiography.

Pflugers Arch. 2011-7-9

[9]
Ghrelin inhibits autonomic function in healthy controls, but has no effect on obese and vagotomized subjects.

Clin Endocrinol (Oxf). 2010-11

[10]
Ghrelin suppresses cardiac sympathetic activity and prevents early left ventricular remodeling in rats with myocardial infarction.

Am J Physiol Heart Circ Physiol. 2008-1

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