Correction: CDP138 silencing inhibits TGF-β/Smad signaling to impair radioresistance and metastasis via GDF15 in lung cancer.
作者信息
Lu Yanwei, Ma Jia, Li Yan, Huang Jing, Zhang Sheng, Yin Zhongyuan, Ren Jinghua, Huang Kai, Wu Gang, Yang Kunyu, Xu Shuangbing
机构信息
Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, China.
Clinic Center of Human Gene Research, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, 430022, China.
出版信息
Cell Death Dis. 2024 Jul 31;15(7):549. doi: 10.1038/s41419-024-06823-2.
Abstract
摘要
相似文献
1
Correction: CDP138 silencing inhibits TGF-β/Smad signaling to impair radioresistance and metastasis via GDF15 in lung cancer.更正：CDP138沉默通过GDF15抑制TGF-β/Smad信号传导，从而损害肺癌的放射抗性和转移。
Cell Death Dis. 2024 Jul 31;15(7):549. doi: 10.1038/s41419-024-06823-2.
2
CDP138 silencing inhibits TGF-β/Smad signaling to impair radioresistance and metastasis via GDF15 in lung cancer.沉默 CDP138 通过 GDF15 抑制 TGF-β/Smad 信号通路以损害肺癌的放射抵抗和转移。
Cell Death Dis. 2017 Sep 7;8(9):e3036. doi: 10.1038/cddis.2017.434.
3
GDF15 contributes to radioresistance and cancer stemness of head and neck cancer by regulating cellular reactive oxygen species via a SMAD-associated signaling pathway.生长分化因子15通过SMAD相关信号通路调节细胞活性氧，从而促进头颈癌的放射抗性和癌干性。
Oncotarget. 2017 Jan 3;8(1):1508-1528. doi: 10.18632/oncotarget.13649.
4
The Upregulation of Caffeic Acid Phenethyl Ester on Growth Differentiation Factor 15 Inhibits Transforming Growth Factor β/Smad Signaling in Bladder Carcinoma Cells.咖啡酸苯乙酯上调生长分化因子15对膀胱癌细胞中转化生长因子β/Smad信号通路的抑制作用
Biomedicines. 2022 Jul 7;10(7):1625. doi: 10.3390/biomedicines10071625.
5
Long non-coding RNA ANRIL promotes the invasion and metastasis of thyroid cancer cells through TGF-β/Smad signaling pathway.长链非编码RNA ANRIL通过TGF-β/Smad信号通路促进甲状腺癌细胞的侵袭和转移。
Oncotarget. 2016 Sep 6;7(36):57903-57918. doi: 10.18632/oncotarget.11087.
6
HOXC6 gene silencing inhibits epithelial-mesenchymal transition and cell viability through the TGF-β/smad signaling pathway in cervical carcinoma cells.HOXC6基因沉默通过TGF-β/smad信号通路抑制宫颈癌细胞的上皮-间质转化和细胞活力。
Cancer Cell Int. 2018 Dec 12;18:204. doi: 10.1186/s12935-018-0680-2. eCollection 2018.
7
KLF17 empowers TGF-β/Smad signaling by targeting Smad3-dependent pathway to suppress tumor growth and metastasis during cancer progression.KLF17通过靶向Smad3依赖性途径增强TGF-β/Smad信号传导，从而在癌症进展过程中抑制肿瘤生长和转移。
Cell Death Dis. 2015 Mar 12;6(3):e1681. doi: 10.1038/cddis.2015.48.
8
Overexpression of Growth Differentiation Factor 15 in Glioblastoma Stem Cells Promotes Their Radioresistance.生长分化因子15在胶质母细胞瘤干细胞中的过表达促进其放射抗性。
Cancers (Basel). 2023 Dec 20;16(1):27. doi: 10.3390/cancers16010027.
9
MicroRNA-527 inhibits TGF-β/SMAD induced epithelial-mesenchymal transition via downregulating SULF2 expression in non-small-cell lung cancer.微小 RNA-527 通过下调非小细胞肺癌中 SULF2 的表达抑制 TGF-β/SMAD 诱导的上皮-间充质转化。
Math Biosci Eng. 2019 May 23;16(5):4607-4621. doi: 10.3934/mbe.2019231.
10
The evolutionarily conserved deubiquitinase UBH1/UCH-L1 augments DAF7/TGF-β signaling, inhibits dauer larva formation, and enhances lung tumorigenesis.进化上保守的去泛素化酶 UBH1/UCH-L1 增强了 DAF7/TGF-β 信号通路，抑制了 dauer 幼虫的形成，并增强了肺癌的发生。
J Biol Chem. 2020 Jul 3;295(27):9105-9120. doi: 10.1074/jbc.RA119.011222. Epub 2020 May 5.
Zotero 插件