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一例巨大髂腰肌脓肿穿孔并与输尿管相通的病例。

A Case of a Huge Iliopsoas Abscess that Perforated and Communicated with the Ureter.

作者信息

Tabata Yusuke, Yanagi Masato, Enomoto Kazuo, Omori Satoshi, Murotani Noriyoshi, Mitsuhashi Osamu, Yasuda Mitsunori, Sawano Yuki, Omori Koichiro, Tabata Yoichiro, Majima Tokifumi

机构信息

Department of Orthopedic Surgery, Mitsuhashi Meisei Hospital, Chiba, Japan.

Hemodialysis Department, Mitsuhashi Meisei Hospital, Chiba, Japan.

出版信息

JMA J. 2024 Jul 16;7(3):449-452. doi: 10.31662/jmaj.2023-0217. Epub 2024 Jun 3.

Abstract

We experienced a rare case in which iliopsoas abscess (IPA), caused by an Extended Spectrum β-Lactamase (ESBL)-producing Proteus mirabilis, perforated and communicated with the ureter and caused sepsis. An 84-year-old woman, bedridden due to sequelae of a cerebral hemorrhage, was brought to our hospital with a chief complaint of fever lasting for 3 weeks. Computed tomography (CT) revealed a huge 180 × 110 × 100 mm IPA in the right iliopsoas muscle. The ureter was also found to communicate with the iliopsoas muscle abscess, ureteral stenosis was detected at the same site, and dilatation of the renal pelvis occurred above the area of the ureteral stenosis, indicating hydronephrosis. Considering the mechanism of this case, if the ureter first ruptures and urine leaks, followed by the formation of an IPA, urine will flow along the surrounding fatty tissue and cause an abscess around the ureter and kidney. However, because almost no abscess was detected around the ureter, the abscess was thought to have originated from the iliopsoas muscle located near the center of the ureter. In summary, in this case, an abscess first formed within the iliopsoas muscle, which gradually expanded and compressed the right ureter, resulting in hydronephrosis. The upper ureter, which had become dilated and thinned due to ureteral obstruction, became even more fragile because of the spread of inflammation from the IPA, and the IPA perforated and communicated with the ureter. In patients who have difficulty communicating, the diagnosis of IPA may be delayed because the only symptom is fever. As in this case, if the diagnosis is delayed, the abscess may become large and perforate the ureter; thus, IPA should always be considered as a cause of fever of unknown origin.

摘要

我们遇到了一例罕见病例,由产超广谱β-内酰胺酶(ESBL)的奇异变形杆菌引起的髂腰肌脓肿(IPA)发生穿孔并与输尿管相通,导致败血症。一名84岁女性因脑出血后遗症卧床,因持续发热3周为主诉被送至我院。计算机断层扫描(CT)显示右侧髂腰肌有一个巨大的180×110×100mm的IPA。还发现输尿管与髂腰肌脓肿相通,在同一部位检测到输尿管狭窄,输尿管狭窄部位上方肾盂扩张,提示肾积水。考虑到该病例的机制,如果输尿管先破裂漏尿,随后形成IPA,尿液将沿周围脂肪组织流动,导致输尿管和肾脏周围形成脓肿。然而,由于输尿管周围几乎未检测到脓肿,因此认为脓肿起源于输尿管中心附近的髂腰肌。总之,在该病例中,脓肿首先在髂腰肌内形成,逐渐扩大并压迫右侧输尿管,导致肾积水。由于输尿管梗阻而扩张变薄的上段输尿管,因IPA炎症扩散而变得更加脆弱,IPA穿孔并与输尿管相通。对于沟通困难的患者,由于唯一症状是发热,IPA的诊断可能会延迟。如此病例所示,如果诊断延迟,脓肿可能会变大并穿破输尿管;因此,IPA应始终被视为不明原因发热的病因之一。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/e1ec/11301075/24f9deba9afb/2433-3298-7-3-0449-g001.jpg

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