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SOX2-Dependent Wound Repair Signature Triggers Prohealing Outcome in Hyperglycemic Wounds.

作者信息

O'Neill Christopher G, Sawaya Andrew P, Mehdizadeh Spencer, Brooks Stephen R, Hasneen Kowser, Nayak Subhashree, Overmiller Andrew M, Morasso Maria I

机构信息

Laboratory of Skin Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland, USA.

Biodata Mining and Discovery Section, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland, USA.

出版信息

J Invest Dermatol. 2025 Feb;145(2):451-455.e5. doi: 10.1016/j.jid.2024.07.012. Epub 2024 Aug 8.

Abstract
摘要

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本文引用的文献

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Intrinsic Networks Regulating Tissue Repair: Comparative Studies of Oral and Skin Wound Healing.
Cold Spring Harb Perspect Biol. 2022 Nov 1;14(11):a041244. doi: 10.1101/cshperspect.a041244.
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A review of granulocyte colony-stimulating factor receptor signaling and regulation with implications for cancer.
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FOXM1 network in association with TREM1 suppression regulates NET formation in diabetic foot ulcers.
EMBO Rep. 2022 Aug 3;23(8):e54558. doi: 10.15252/embr.202154558. Epub 2022 Jul 20.
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Deregulated immune cell recruitment orchestrated by FOXM1 impairs human diabetic wound healing.
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SOX2 Epidermal Overexpression Promotes Cutaneous Wound Healing via Activation of EGFR/MEK/ERK Signaling Mediated by EGFR Ligands.
J Invest Dermatol. 2019 Aug;139(8):1809-1820.e8. doi: 10.1016/j.jid.2019.02.004. Epub 2019 Feb 14.
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Transcriptional signature primes human oral mucosa for rapid wound healing.
Sci Transl Med. 2018 Jul 25;10(451). doi: 10.1126/scitranslmed.aap8798.
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Diabetic Foot Ulcers and Epidermal Growth Factor: Revisiting the Local Delivery Route for a Successful Outcome.
Biomed Res Int. 2017;2017:2923759. doi: 10.1155/2017/2923759. Epub 2017 Aug 21.
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The Ca2+-binding proteins S100A8 and S100A9 are encoded by novel injury-regulated genes.
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