Retraction: 20-HETE inhibition by HET0016 decreases the blood-brain barrier permeability and brain edema after traumatic brain injury.
出版信息
Front Aging Neurosci. 2024 Jul 29;16:1469759. doi: 10.3389/fnagi.2024.1469759. eCollection 2024.
Abstract
[This retracts the article DOI: 10.3389/fnagi.2018.00207.].
摘要
相似文献
1
Retraction: 20-HETE inhibition by HET0016 decreases the blood-brain barrier permeability and brain edema after traumatic brain injury.
Front Aging Neurosci. 2024 Jul 29;16:1469759. doi: 10.3389/fnagi.2024.1469759. eCollection 2024.
2
Retraction: Corrigendum: 20-HETE inhibition by HET0016 decreases the blood-brain barrier permeability and brain edema after traumatic brain injury.
Front Aging Neurosci. 2024 Jul 29;16:1469763. doi: 10.3389/fnagi.2024.1469763. eCollection 2024.
3
Corrigendum: 20-HETE Inhibition by HET0016 Decreases the Blood-Brain Barrier Permeability and Brain Edema After Traumatic Brain Injury.勘误：HET0016对20-羟基二十碳四烯酸的抑制作用可降低创伤性脑损伤后的血脑屏障通透性和脑水肿。
Front Aging Neurosci. 2018 Aug 20;10:239. doi: 10.3389/fnagi.2018.00239. eCollection 2018.
4
20-HETE Inhibition by HET0016 Decreases the Blood-Brain Barrier Permeability and Brain Edema After Traumatic Brain Injury.HET0016抑制20-HETE可降低创伤性脑损伤后的血脑屏障通透性和脑水肿。
Front Aging Neurosci. 2018 Jul 16;10:207. doi: 10.3389/fnagi.2018.00207. eCollection 2018.
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The protective effect of HET0016 on brain edema and blood-brain barrier dysfunction after cerebral ischemia/reperfusion.HET0016对脑缺血/再灌注后脑水肿和血脑屏障功能障碍的保护作用。
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Reactive oxygen species-responsive HET0016 prodrug-loaded liposomes attenuate neuroinflammation and improve neurological deficit in a rat model of juvenile traumatic brain injury.活性氧物种响应性负载HET0016前药的脂质体减轻幼年创伤性脑损伤大鼠模型中的神经炎症并改善神经功能缺损
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Intravenous formulation of N-hydroxy-N'-(4-n-butyl-2-methylphenyl)formamidine (HET0016) for inhibition of rat brain 20-hydroxyeicosatetraenoic acid formation.用于抑制大鼠脑内20-羟基二十碳四烯酸生成的N-羟基-N'-(4-正丁基-2-甲基苯基)甲脒（HET0016）静脉制剂
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Protective effect of the 20-HETE inhibitor HET0016 on brain damage after temporary focal ischemia.20-羟基二十碳四烯酸（20-HETE）抑制剂HET0016对短暂性局灶性缺血后脑损伤的保护作用。
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20-HETE synthesis inhibition attenuates traumatic brain injury-induced mitochondrial dysfunction and neuronal apoptosis via the SIRT1/PGC-1α pathway: A translational study.20-HETE 合成抑制通过 SIRT1/PGC-1α 通路减轻创伤性脑损伤诱导的线粒体功能障碍和神经元凋亡：一项转化研究。
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Administration of a 20-Hydroxyeicosatetraenoic Acid Synthesis Inhibitor Improves Outcome in a Rat Model of Pediatric Traumatic Brain Injury.20-羟二十碳四烯酸合成抑制剂的给药可改善小儿创伤性脑损伤大鼠模型的预后。
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