勘误：动脉粥样硬化中血管平滑肌细胞与巨噬细胞之间的分子相互作用
Corrigendum: Molecular interactions between vascular smooth muscle cells and macrophages in atherosclerosis.
作者信息
Beck-Joseph Jahnic, Tabrizian Maryam, Lehoux Stephanie
机构信息
Biomat'X Research Laboratories, Department of Biomedical Engineering, McGill University, Montreal, QC, Canada.
Department of Medicine, Lady Davis Institute, McGill University, Montreal, QC, Canada.
出版信息
Front Cardiovasc Med. 2024 Aug 12;11:1462284. doi: 10.3389/fcvm.2024.1462284. eCollection 2024.
Abstract
[This corrects the article DOI: 10.3389/fcvm.2021.737934.].
摘要
[本文更正了文章的数字对象标识符：10.3389/fcvm.2021.737934。]
相似文献
1
Corrigendum: Molecular interactions between vascular smooth muscle cells and macrophages in atherosclerosis.勘误：动脉粥样硬化中血管平滑肌细胞与巨噬细胞之间的分子相互作用
Front Cardiovasc Med. 2024 Aug 12;11:1462284. doi: 10.3389/fcvm.2024.1462284. eCollection 2024.
2
Corrigendum: The dual role of low-density lipoprotein receptor-related protein 1 in atherosclerosis.勘误：低密度脂蛋白受体相关蛋白1在动脉粥样硬化中的双重作用。
Front Cardiovasc Med. 2023 Apr 21;10:1179643. doi: 10.3389/fcvm.2023.1179643. eCollection 2023.
3
Molecular Interactions Between Vascular Smooth Muscle Cells and Macrophages in Atherosclerosis.动脉粥样硬化中血管平滑肌细胞与巨噬细胞之间的分子相互作用
Front Cardiovasc Med. 2021 Oct 15;8:737934. doi: 10.3389/fcvm.2021.737934. eCollection 2021.
4
Corrigendum: Atherogenic index of plasma and coronary artery disease in the adult population: A meta-analysis.勘误：成人血浆致动脉粥样硬化指数与冠状动脉疾病：一项荟萃分析。
Front Cardiovasc Med. 2023 Mar 8;10:1153914. doi: 10.3389/fcvm.2023.1153914. eCollection 2023.
5
Molecular and functional interactions among monocytes/macrophages and smooth muscle cells and their relevance for atherosclerosis.单核细胞/巨噬细胞与平滑肌细胞之间的分子和功能相互作用及其与动脉粥样硬化的相关性。
Crit Rev Eukaryot Gene Expr. 2014;24(4):341-55. doi: 10.1615/critreveukaryotgeneexpr.2014012157.
6
The Gene at the 15q26 Coronary-Artery-Disease Locus Inhibits Atherosclerosis.15q26 号染色体冠状动脉疾病基因抑制动脉粥样硬化。
Circ Res. 2022 Dec 2;131(12):1004-1017. doi: 10.1161/CIRCRESAHA.122.321146. Epub 2022 Nov 2.
7
Neopterin Counters Vascular Inflammation and Atherosclerosis.新蝶呤可对抗血管炎症和动脉粥样硬化。
J Am Heart Assoc. 2018 Feb 2;7(3):e007359. doi: 10.1161/JAHA.117.007359.
8
The roles of mechanotransduction, vascular wall cells, and blood cells in atheroma induction.机械转导、血管壁细胞和血细胞在动脉粥样硬化形成中的作用。
Vascular. 2019 Feb;27(1):98-109. doi: 10.1177/1708538118796063. Epub 2018 Aug 29.
9
Macrophages regulate vascular smooth muscle cell function during atherosclerosis progression through IL-1β/STAT3 signaling.巨噬细胞通过 IL-1β/STAT3 信号通路调节动脉粥样硬化进程中的血管平滑肌细胞功能。
Commun Biol. 2022 Dec 1;5(1):1316. doi: 10.1038/s42003-022-04255-2.
10
NLRP3 Inflammasome Activation Controls Vascular Smooth Muscle Cells Phenotypic Switch in Atherosclerosis.NLRP3 炎性小体激活调控动脉粥样硬化中血管平滑肌细胞表型转换。
Int J Mol Sci. 2021 Dec 29;23(1):340. doi: 10.3390/ijms23010340.
引用本文的文献
1
The Atherosclerotic Plaque Microenvironment as a Therapeutic Target.作为治疗靶点的动脉粥样硬化斑块微环境
Curr Atheroscler Rep. 2025 Apr 2;27(1):47. doi: 10.1007/s11883-025-01294-y.
Zotero 插件