• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

勘误：细胞外HSP90α与内质网应激相互作用，通过PI3K/AKT途径促进肺纤维化中肺成纤维细胞的激活。

Corrigendum: Extracellular HSP90α interacts with ER stress to promote fibroblasts activation through PI3K/AKT pathway in pulmonary fibrosis.

作者信息

Zhang Jinming, Zhong Wenshan, Liu Yuanyuan, Chen Weimou, Lu Ye, Zeng Zhaojin, Qiao Yujie, Huang Haohua, Wan Xuan, Li Wei, Meng Xiaojing, Zou Fei, Cai Shaoxi, Dong Hangming

机构信息

Chronic Airways Diseases Laboratory, Department of Respiratory and Critical Care Medicine, Nanfang Hospital, Southern Medical University, Guangzhou, China.

Department of Dermatology and The Norris Comprehensive Cancer Centre, University of Southern California Keck Medical Centre, Los Angeles, CA, United States.

出版信息

Front Pharmacol. 2024 Aug 12;15:1296214. doi: 10.3389/fphar.2024.1296214. eCollection 2024.

DOI:10.3389/fphar.2024.1296214
PMID:39188948
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11345549/
Abstract

[This corrects the article DOI: 10.3389/fphar.2021.708462.].

摘要

[本文更正了文章的数字对象标识符：10.3389/fphar.2021.708462。]

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d1fa/11345549/92bbd42ea41d/fphar-15-1296214-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d1fa/11345549/4c7d1e12acbb/fphar-15-1296214-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d1fa/11345549/92bbd42ea41d/fphar-15-1296214-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d1fa/11345549/4c7d1e12acbb/fphar-15-1296214-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d1fa/11345549/92bbd42ea41d/fphar-15-1296214-g007.jpg

相似文献

1
Corrigendum: Extracellular HSP90α interacts with ER stress to promote fibroblasts activation through PI3K/AKT pathway in pulmonary fibrosis.勘误：细胞外HSP90α与内质网应激相互作用，通过PI3K/AKT途径促进肺纤维化中肺成纤维细胞的激活。
Front Pharmacol. 2024 Aug 12;15:1296214. doi: 10.3389/fphar.2024.1296214. eCollection 2024.
2
Extracellular HSP90α Interacts With ER Stress to Promote Fibroblasts Activation Through PI3K/AKT Pathway in Pulmonary Fibrosis.细胞外HSP90α与内质网应激相互作用，通过PI3K/AKT途径促进肺纤维化中成纤维细胞的激活。
Front Pharmacol. 2021 Aug 23;12:708462. doi: 10.3389/fphar.2021.708462. eCollection 2021.
3
Involvement of ER stress, PI3K/AKT activation, and lung fibroblast proliferation in bleomycin-induced pulmonary fibrosis.博来霉素诱导肺纤维化中内质网应激、PI3K/AKT 激活和肺成纤维细胞增殖的作用。
Sci Rep. 2017 Oct 27;7(1):14272. doi: 10.1038/s41598-017-14612-5.
4
Corrigendum: DT-13 Inhibits Proliferation and Metastasis of Human Prostate Cancer Cells Through Blocking PI3K/Akt Pathway.勘误：DT - 13通过阻断PI3K/Akt通路抑制人前列腺癌细胞的增殖和转移。
Front Pharmacol. 2020 Apr 8;11:462. doi: 10.3389/fphar.2020.00462. eCollection 2020.
5
Corrigendum: Isorhamnetin Inhibits Human Gallbladder Cancer Cell Proliferation and Metastasis PI3K/AKT Signaling Pathway Inactivation.勘误：异鼠李素通过PI3K/AKT信号通路失活抑制人胆囊癌细胞增殖和转移。
Front Pharmacol. 2021 Oct 20;12:792330. doi: 10.3389/fphar.2021.792330. eCollection 2021.
6
Corrigendum: Xiaoyaosan alleviates hippocampal glutamate-induced toxicity in the CUMS rats via NR2B and PI3K/Akt signaling pathway.勘误：逍遥散通过NR2B和PI3K/Akt信号通路减轻慢性不可预知温和应激（CUMS）大鼠海马谷氨酸诱导的毒性。
Front Pharmacol. 2022 Dec 15;13:1042762. doi: 10.3389/fphar.2022.1042762. eCollection 2022.
7
Corrigendum: Cinnamaldehyde suppressed EGF-induced EMT process and inhibits ovarian cancer progression through PI3K/AKT pathway.勘误：肉桂醛通过PI3K/AKT途径抑制表皮生长因子诱导的上皮-间质转化过程并抑制卵巢癌进展。
Front Pharmacol. 2024 Jul 8;15:1427330. doi: 10.3389/fphar.2024.1427330. eCollection 2024.
8
Neotuberostemonine and tuberostemonine ameliorate pulmonary fibrosis through suppressing TGF-β and SDF-1 secreted by macrophages and fibroblasts via the PI3K-dependent AKT and ERK pathways.新土木香内酯和土木香内酯通过抑制巨噬细胞和成纤维细胞分泌的 TGF-β和 SDF-1，通过 PI3K 依赖性 AKT 和 ERK 通路改善肺纤维化。
Chin J Nat Med. 2023 Jul;21(7):527-539. doi: 10.1016/S1875-5364(23)60444-3.
9
Corrigendum: Neferine Ameliorates Sepsis-Induced Myocardial Dysfunction Through Anti-Apoptotic and Antioxidative Effects by Regulating the PI3K/AKT/mTOR Signaling Pathway.勘误：莲心碱通过调节PI3K/AKT/mTOR信号通路的抗凋亡和抗氧化作用改善脓毒症诱导的心肌功能障碍。
Front Pharmacol. 2022 Apr 29;13:913778. doi: 10.3389/fphar.2022.913778. eCollection 2022.
10
Eupatilin inhibits pulmonary fibrosis by activating Sestrin2/PI3K/Akt/mTOR dependent autophagy pathway.柚皮苷通过激活 Sestrin2/PI3K/Akt/mTOR 依赖性自噬途径抑制肺纤维化。
Life Sci. 2023 Dec 1;334:122218. doi: 10.1016/j.lfs.2023.122218. Epub 2023 Nov 1.