Toll-like Receptor 9 in Preclinical Models of Idiopathic Pulmonary Fibrosis.
作者信息
Verma Priyanka, Hamill Timothy, Bhattacharyya Swati
机构信息
University of Michigan, Ann Arbor, Michigan, United States.
University of Michigan-Ann Arbor, Internal medicine, Ann Arbor, Michigan, United States;
出版信息
Am J Respir Crit Care Med. 2024 Sep 25;211(1):9-10. doi: 10.1164/rccm.202408-1578ED.
Abstract
摘要
相似文献
1
Toll-like Receptor 9 in Preclinical Models of Idiopathic Pulmonary Fibrosis.特发性肺纤维化临床前模型中的Toll样受体9
Am J Respir Crit Care Med. 2024 Sep 25;211(1):9-10. doi: 10.1164/rccm.202408-1578ED.
2
Disparate Interferon Signaling and Shared Aberrant Basaloid Cells in Single-Cell Profiling of Idiopathic Pulmonary Fibrosis and Systemic Sclerosis-Associated Interstitial Lung Disease.特发性肺纤维化和系统性硬化症相关间质性肺疾病单细胞分析中的不同干扰素信号传导和共享异常基底样细胞
Front Immunol. 2021 Mar 30;12:595811. doi: 10.3389/fimmu.2021.595811. eCollection 2021.
3
Prognosis after acute exacerbation in patients with interstitial lung disease other than idiopathic pulmonary fibrosis.特发性肺纤维化以外的间质性肺疾病患者急性加重后的预后。
Clin Respir J. 2021 Mar;15(3):336-344. doi: 10.1111/crj.13304. Epub 2020 Dec 10.
4
Plasma LTBP2 as a potential biomarker in differential diagnosis of connective tissue disease-associated interstitial lung disease and idiopathic pulmonary fibrosis: a pilot study.血浆 LTBP2 作为结缔组织病相关间质性肺疾病和特发性肺纤维化鉴别诊断的潜在生物标志物：一项初步研究。
Clin Exp Med. 2023 Dec;23(8):4809-4816. doi: 10.1007/s10238-023-01214-x. Epub 2023 Oct 21.
5
Plasma Surfactant Protein-D, Matrix Metalloproteinase-7, and Osteopontin Index Distinguishes Idiopathic Pulmonary Fibrosis from Other Idiopathic Interstitial Pneumonias.血浆表面活性蛋白-D、基质金属蛋白酶-7和骨桥蛋白指数可将特发性肺纤维化与其他特发性间质性肺炎区分开来。
Am J Respir Crit Care Med. 2016 Nov 15;194(10):1242-1251. doi: 10.1164/rccm.201505-0862OC.
6
Hospitalization Rates in Interstitial Lung Disease: An Analysis of the Pulmonary Fibrosis Foundation Registry.间质性肺疾病的住院率：对肺纤维化基金会登记处的分析。
Am J Respir Crit Care Med. 2024 Sep 15;210(6):801-813. doi: 10.1164/rccm.202309-1708OC.
7
Family History of Pulmonary Fibrosis Predicts Worse Survival in Patients With Interstitial Lung Disease.特发性肺纤维化家族史预示着特发性间质性肺病患者的生存预后更差。
Chest. 2021 May;159(5):1913-1921. doi: 10.1016/j.chest.2021.01.026. Epub 2021 Jan 21.
8
Claims-based Prevalence of Disease Progression among Patients with Fibrosing Interstitial Lung Disease Other than Idiopathic Pulmonary Fibrosis in the United States.美国非特发性肺纤维化纤维化性间质性肺疾病患者疾病进展的基于索赔的患病率。
Ann Am Thorac Soc. 2022 Jul;19(7):1112-1121. doi: 10.1513/AnnalsATS.202102-222OC.
9
Smoking-related emphysema is associated with idiopathic pulmonary fibrosis and rheumatoid lung.与吸烟有关的肺气肿与特发性肺纤维化和类风湿肺有关。
Respirology. 2013 Nov;18(8):1191-6. doi: 10.1111/resp.12154.
10
Healthcare Resources Utilization and Costs of Patients with Non-IPF Progressive Fibrosing Interstitial Lung Disease Based on Insurance Claims in the USA.基于美国保险索赔数据的非特发性肺纤维化进行性纤维化性间质性肺疾病患者的医疗资源利用和费用。
Adv Ther. 2020 Jul;37(7):3292-3298. doi: 10.1007/s12325-020-01380-4. Epub 2020 May 21.
本文引用的文献
1
Toll-like Receptor 9 Inhibition Mitigates Fibroproliferative Responses in Translational Models of Pulmonary Fibrosis.Toll样受体9抑制减轻肺纤维化转化模型中的纤维增生反应。
Am J Respir Crit Care Med. 2024 Aug 27;211(1):91-102. doi: 10.1164/rccm.202401-0065OC.
2
Toll-Like Receptor 9 Aggravates Pulmonary Fibrosis by Promoting NLRP3-Mediated Pyroptosis of Alveolar Epithelial Cells.Toll 样受体 9 通过促进肺泡上皮细胞 NLRP3 介导的焦亡加重肺纤维化。
Inflammation. 2024 Oct;47(5):1744-1761. doi: 10.1007/s10753-024-02006-5. Epub 2024 Mar 18.
3
Mitochondrial DNA Sensing Pathogen Recognition Receptors in Systemic Sclerosis Associated Interstitial Lung Disease: A Review.系统性硬化症相关间质性肺病中线粒体DNA传感病原体识别受体：综述
Curr Treatm Opt Rheumatol. 2023 Dec;9(4):204-220. doi: 10.1007/s40674-023-00211-1. Epub 2023 Aug 8.
4
Fibronectin-EDA accumulates via reduced ubiquitination downstream of Toll-like receptor 9 activation in SSc-ILD fibroblasts.纤维连接蛋白-EDA 通过 Toll 样受体 9 激活下游的泛素化减少在 SSc-ILD 成纤维细胞中积累。
Am J Physiol Lung Cell Mol Physiol. 2022 Oct 1;323(4):L484-L494. doi: 10.1152/ajplung.00019.2022. Epub 2022 Aug 23.
5
Idiopathic pulmonary fibrosis.特发性肺纤维化。
Nat Rev Dis Primers. 2017 Oct 20;3:17074. doi: 10.1038/nrdp.2017.74.
6
Toll-like receptors in the pathogenesis of pulmonary fibrosis.Toll样受体在肺纤维化发病机制中的作用
Eur J Pharmacol. 2017 Aug 5;808:35-43. doi: 10.1016/j.ejphar.2016.06.045. Epub 2016 Jun 27.
7
Toll-like Receptor 9 Signaling Is Augmented in Systemic Sclerosis and Elicits Transforming Growth Factor β-Dependent Fibroblast Activation.Toll 样受体 9 信号在系统性硬化症中增强，并引发转化生长因子 β 依赖性成纤维细胞激活。
Arthritis Rheumatol. 2016 Aug;68(8):1989-2002. doi: 10.1002/art.39655.
8
Sustained activation of toll-like receptor 9 induces an invasive phenotype in lung fibroblasts: possible implications in idiopathic pulmonary fibrosis.Toll样受体9的持续激活诱导肺成纤维细胞出现侵袭性表型：对特发性肺纤维化的潜在影响
Am J Pathol. 2015 Apr;185(4):943-57. doi: 10.1016/j.ajpath.2014.12.011. Epub 2015 Feb 7.
9
TLR9 differentiates rapidly from slowly progressing forms of idiopathic pulmonary fibrosis.TLR9 可迅速从特发性肺纤维化的缓慢进展形式中分化出来。
Sci Transl Med. 2010 Nov 10;2(57):57ra82. doi: 10.1126/scitranslmed.3001510.
Zotero 插件