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鉴定与 2W 型腓骨肌萎缩症相关的组氨酰-tRNA 合成酶基因中的一种新型杂合变异。

Characterization of a novel heterozygous variant in the histidyl-tRNA synthetase gene associated with Charcot-Marie-Tooth disease type 2W.

机构信息

Department of Biochemistry, The University of Western Ontario, London, Ontario, Canada.

Division of Medical Genetics, Department of Paediatrics, The University of Western Ontario, London, Ontario, Canada.

出版信息

IUBMB Life. 2024 Dec;76(12):1125-1138. doi: 10.1002/iub.2918. Epub 2024 Oct 1.

DOI:10.1002/iub.2918
PMID:39352000
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11580374/
Abstract

Heterozygous pathogenic variants in the histidyl-tRNA synthetase (HARS) gene are associated with Charcot-Marie-Tooth (CMT) type 2W disease, classified as an axonal peripheral neuropathy. To date, at least 60 variants causing CMT symptoms have been identified in seven different aminoacyl-tRNA synthetases, with eight being found in the catalytic domain of HARS. The genetic data clearly show a causative role of aminoacyl-tRNA synthetases in CMT; however, the cellular mechanisms leading to pathology can vary widely and are unknown in the case of most identified variants. Here we describe a novel HARS variant, c.412T>C; p.Y138H, identified through a CMT gene panel in a patient with peripheral neuropathy. To determine the effect of p.Y138H we employed a humanized HARS yeast model and recombinant protein biochemistry, which identified a deficiency in protein dimerization and a growth defect which shows mild but significant improvement with histidine supplementation. This raises the potential for a clinical trial of histidine.

摘要

杂合致病性变异位于组氨酰-tRNA 合成酶(HARS)基因中,与 2W 型腓骨肌萎缩症(Charcot-Marie-Tooth,CMT)相关,归类为轴索性周围神经病。迄今为止,已在七种不同的氨酰-tRNA 合成酶中发现了至少 60 种导致 CMT 症状的变异,其中 8 种位于 HARS 的催化结构域中。遗传数据清楚地表明氨酰-tRNA 合成酶在 CMT 中起因果作用;然而,导致病理的细胞机制可能差异很大,而且大多数已确定的变异都未知。在这里,我们描述了一种通过患者周围神经病变的 CMT 基因面板鉴定的新型 HARS 变体 c.412T>C;p.Y138H。为了确定 p.Y138H 的影响,我们使用了人源化 HARS 酵母模型和重组蛋白生物化学,发现其蛋白二聚体缺陷和生长缺陷,而补充组氨酸则显示出轻度但显著的改善。这就提出了进行组氨酸临床试验的可能性。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/ad028f150d3b/IUB-76-1125-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/11fac8b9068b/IUB-76-1125-g006.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/24c43c813c16/IUB-76-1125-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/ad028f150d3b/IUB-76-1125-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/11fac8b9068b/IUB-76-1125-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/8a009e5086d3/IUB-76-1125-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/870f440b5dd1/IUB-76-1125-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/24c43c813c16/IUB-76-1125-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/320e/11580374/ad028f150d3b/IUB-76-1125-g005.jpg

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本文引用的文献

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Dominant mutations causing axonal Charcot-Marie-Tooth disease expand -associated diseases.导致轴索性夏科-马里-图思病的显性突变会引发相关疾病。
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Towards a Cure for HARS Disease.为治疗 HARS 疾病而努力。
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