核因子κB抑制肺上皮细胞中视黄酸受体介导的GPRC5A反式激活，从而促进肿瘤形成。 - Suppr | 超能文献

NF-κB represses retinoic acid receptor-mediated GPRC5A transactivation in lung epithelial cells to promote neoplasia.

作者信息

Song Hongyong, Ye Xiaofeng, Liao Yueling, Zhang Siwei, Xu Dongliang, Zhong Shuangshuang, Jing Bo, Wang Tong, Sun Beibei, Xu Jianhua, Guo Wenzheng, Li Kaimi, Hu Min, Kuang Yanbin, Ling Jing, Zhang Tuo, Wu Yadi, Du Jing, Yao Feng, Chin Y Eugene, Wang Qi, Zhou Binhua P, Deng Jiong

出版信息

JCI Insight. 2024 Oct 8;9(19):e186558. doi: 10.1172/jci.insight.186558.

DOI:10.1172/jci.insight.186558

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11466182/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c5b8/11466182/6b1df7afca70/jciinsight-9-186558-g134.jpg

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本文引用的文献

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Lung Tumor Suppressor GPRC5A Binds EGFR and Restrains Its Effector Signaling.肺肿瘤抑制因子 GPRC5A 与 EGFR 结合并抑制其效应信号转导。

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