海胆胚胎原肠胚形成对紫外线抑制的双相阶段敏感性。
Biphasic stage sensitivity to UV suppression of gastrulation in sea urchin embryos.
作者信息
Amemiya S, Yonemura S, Kinoshita S, Shiroya T
出版信息
Cell Differ. 1986 Jan;18(1):45-9. doi: 10.1016/0045-6039(86)90034-5.
PMID:3940661
Abstract
The effects of ultraviolet light (UV) on the gastrulation of sea urchin embryos were examined. The results suggest that gastrulation is inhibited by UV irradiation and that stage sensitivity to UV suppression of gastrulation changes biphasically: higher sensitivity at early and late blastula, and lower sensitivity at the mid-blastula stages. The UV-induced inhibition of gastrulation was completely reversible by subsequent exposure to visible light.
摘要
研究了紫外线（UV）对海胆胚胎原肠胚形成的影响。结果表明，紫外线照射会抑制原肠胚形成，并且原肠胚形成对紫外线抑制的阶段敏感性呈双相变化：在囊胚早期和晚期敏感性较高，而在囊胚中期敏感性较低。紫外线诱导的原肠胚形成抑制在随后暴露于可见光后可完全逆转。
相似文献
1
Biphasic stage sensitivity to UV suppression of gastrulation in sea urchin embryos.海胆胚胎原肠胚形成对紫外线抑制的双相阶段敏感性。
Cell Differ. 1986 Jan;18(1):45-9. doi: 10.1016/0045-6039(86)90034-5.
2
Ultraviolet irradiation during ooplasmic segregation prevents gastrulation, sensory cell induction, and axis formation in the ascidian embryo.卵质分离期间的紫外线照射会阻止海鞘胚胎的原肠胚形成、感觉细胞诱导和轴形成。
Dev Biol. 1990 Aug;140(2):388-400. doi: 10.1016/0012-1606(90)90088-z.
3
Ultraviolet irradiation of eggs and blastomere isolation experiments suggest that gastrulation in the direct developing ascidian, Molgula pacifica, requires localized cytoplasmic determinants in the egg and cell signaling beginning at the two-cell stage.对卵子进行紫外线照射以及卵裂球分离实验表明，在直接发育的海鞘太平洋海鞘中，原肠胚形成需要卵子中局部化的细胞质决定因子以及从两细胞阶段开始的细胞信号传导。
Evol Dev. 2004 May-Jun;6(3):180-6. doi: 10.1111/j.1525-142X.2004.04023.x.
4
An altered series of ectodermal gene expressions accompanying the reversible suspension of differentiation in the zinc-animalized sea urchin embryo.在锌诱导动物化的海胆胚胎中，伴随分化可逆性暂停的一系列外胚层基因表达变化。
Dev Biol. 1986 Mar;114(1):214-24. doi: 10.1016/0012-1606(86)90397-0.
5
Developmental expression of cell-surface (glyco)proteins involved in gastrulation and spicule formation in sea urchin embryos.海胆胚胎中参与原肠胚形成和骨针形成的细胞表面（糖）蛋白的发育表达。
Cold Spring Harb Symp Quant Biol. 1985;50:91-8. doi: 10.1101/sqb.1985.050.01.013.
6
Role for platelet-derived growth factor-like and epidermal growth factor-like signaling pathways in gastrulation and spiculogenesis in the Lytechinus sea urchin embryo.血小板衍生生长因子样和表皮生长因子样信号通路在长海胆胚胎原肠胚形成和骨针形成中的作用。
Dev Dyn. 1995 Sep;204(1):77-88. doi: 10.1002/aja.1002040110.
7
Gastrulation in the sea urchin, Strongylocentrotus purpuratus, is disrupted by the small laminin peptides YIGSR and IKVAV.紫球海胆（Strongylocentrotus purpuratus）的原肠胚形成会被小层粘连蛋白肽YIGSR和IKVAV破坏。
Cell Adhes Commun. 1995 May;3(2):163-77. doi: 10.3109/15419069509081284.
8
Micromere descendants at the blastula stage are involved in normal archenteron formation in sea urchin embryos.囊胚期的小分裂球后代参与海胆胚胎正常原肠的形成。
Dev Genes Evol. 2001 Feb;211(2):83-8. doi: 10.1007/s004270000120.
9
Carbohydrate involvement in cellular interactions in sea urchin gastrulation.碳水化合物在海胆原肠胚形成过程中的细胞相互作用中的作用。
Acta Histochem. 2004;106(2):97-106. doi: 10.1016/j.acthis.2004.01.001.
10
Inhibition of mitogen activated protein kinase signaling affects gastrulation and spiculogenesis in the sea urchin embryo.有丝分裂原活化蛋白激酶信号传导的抑制会影响海胆胚胎的原肠胚形成和骨针发生。
Dev Growth Differ. 2003 Oct-Dec;45(5-6):527-42. doi: 10.1111/j.1440-169x.2003.00710.x.
Zotero 插件