更正：雷瓜等人。TrkA与STAT3相互作用并使其磷酸化，以增强基因转录并促进三阴性和HER2富集型乳腺癌中的乳腺癌干细胞。2021年，，2340。
Correction: Regua et al. TrkA Interacts with and Phosphorylates STAT3 to Enhance Gene Transcription and Promote Breast Cancer Stem Cells in Triple-Negative and HER2-Enriched Breast Cancers. 2021, , 2340.
作者信息
Regua Angelina T, Aguayo Noah R, Jalboush Sara Abu, Doheny Daniel L, Manore Sara G, Zhu Dongqin, Wong Grace L, Arrigo Austin, Wagner Calvin J, Yu Yang, Thomas Alexandra, Chan Michael D, Ruiz Jimmy, Jin Guangxu, Strowd Roy, Sun Peiqing, Lin Jiayuh, Lo Hui-Wen
机构信息
Department of Cancer Biology, Wake Forest University School of Medicine, Winston-Salem, NC 27101, USA.
Department of Hematology and Oncology, Wake Forest University School of Medicine, Winston-Salem, NC 27101, USA.
出版信息
Cancers (Basel). 2024 Oct 8;16(19):3409. doi: 10.3390/cancers16193409.
Abstract
In the original publication [...].
摘要
在原始出版物中 [...]。
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Correction: Regua et al. TrkA Interacts with and Phosphorylates STAT3 to Enhance Gene Transcription and Promote Breast Cancer Stem Cells in Triple-Negative and HER2-Enriched Breast Cancers. 2021, , 2340.更正：雷瓜等人。TrkA与STAT3相互作用并使其磷酸化，以增强基因转录并促进三阴性和HER2富集型乳腺癌中的乳腺癌干细胞。2021年，，2340。
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本文引用的文献
1
TrkA Interacts with and Phosphorylates STAT3 to Enhance Gene Transcription and Promote Breast Cancer Stem Cells in Triple-Negative and HER2-Enriched Breast Cancers.TrkA与STAT3相互作用并使其磷酸化，以增强基因转录并促进三阴性和HER2富集型乳腺癌中的乳腺癌干细胞。
Cancers (Basel). 2021 May 12;13(10):2340. doi: 10.3390/cancers13102340.
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