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中间支闭塞导致的短暂性左束支传导阻滞诱发急性心力衰竭

Transient Left Bundle Branch Block Induced Acute Heart Failure as a Consequence of Ramus Intermedius Occlusion.

作者信息

Eren Mehmet, Velibey Yalcin

机构信息

Department of Cardiology, Siyami Ersek Thoracic and Cardiovascular Surgery Center, Training and Research Hospital, Istanbul, Türkiye.

出版信息

Ann Noninvasive Electrocardiol. 2025 Jan;30(1):e70038. doi: 10.1111/anec.70038.

DOI:10.1111/anec.70038
PMID:39645601
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11625385/
Abstract

We present a case of acute decompensated heart failure due to transient left bundle branch block (LBBB) as a consequence of a ramus intermedius (RI) occlusion. A 63-year-old woman was admitted with acute onset of typical chest pain and pulmonary edema, a newly diagnosed LBBB and left ventricular (LV) systolic dysfunction. A coronary angiogram demonstrated critical stenosis at the osteal segment of RI. After successful coronary revascularization, LBBB was completely resolved, with an improvement in LV systolic function and symptoms. In terms of anatomical blood supply of the left bundle branch, transient LBBB as a consequence of an RI occlusion is unusual.

摘要

我们报告一例因中间支（RI）闭塞导致短暂性左束支传导阻滞（LBBB）引起的急性失代偿性心力衰竭病例。一名63岁女性因典型胸痛和肺水肿急性发作入院，新诊断为LBBB和左心室（LV）收缩功能障碍。冠状动脉造影显示RI起始段严重狭窄。成功进行冠状动脉血运重建后，LBBB完全消失，LV收缩功能和症状改善。就左束支的解剖血供而言，RI闭塞导致的短暂性LBBB并不常见。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/beef/11625385/0cfd61e303e2/ANEC-30-e70038-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/beef/11625385/4e2d1147121b/ANEC-30-e70038-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/beef/11625385/0cfd61e303e2/ANEC-30-e70038-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/beef/11625385/4e2d1147121b/ANEC-30-e70038-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/beef/11625385/0cfd61e303e2/ANEC-30-e70038-g002.jpg

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Electrocardiographic Predictors of Mortality in Acute Anterior Wall Myocardial Infarction With Right Bundle Branch Block and Right Precordial Q-Waves (qRBBB).急性前壁心肌梗死合并右束支阻滞和右胸前导联 Q 波（qRBBB）患者的心电图预测死亡率。
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Transient left bundle branch block and intraventricular dyssynchrony as a cause of reversible left ventricular dysfunction: The "in vivo" documentation of spontaneous electrical remodeling.
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