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Author Correction: GLCCI1 alleviates GRP78-initiated endoplasmic reticulum stress-induced apoptosis of retinal ganglion cells in diabetic retinopathy by upregulating and interacting with HSP90AB1.作者更正：GLCCI1 通过上调 HSP90AB1 并与之相互作用，减轻糖尿病视网膜病变中 GRP78 引发的内质网应激诱导的视网膜神经节细胞凋亡。
Sci Rep. 2024 Dec 16;14(1):30500. doi: 10.1038/s41598-024-83001-6.
2
GLCCI1 alleviates GRP78-initiated endoplasmic reticulum stress-induced apoptosis of retinal ganglion cells in diabetic retinopathy by upregulating and interacting with HSP90AB1.葡萄糖调节蛋白 78 诱导的内质网应激诱导糖尿病视网膜病变中视网膜神经节细胞凋亡，通过上调和相互作用热休克蛋白 90AB1 减轻。
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Neuritin Alleviates Diabetic Retinopathy by Regulating Endoplasmic Reticulum Stress in Rats.神经黏附素通过调节内质网应激缓解大鼠糖尿病视网膜病变。
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Neuroprotective effect of He-Ying-Qing-Re formula on retinal ganglion cell in diabetic retinopathy.和营清热方对糖尿病视网膜病变大鼠视网膜神经节细胞的保护作用。
J Ethnopharmacol. 2018 Mar 25;214:179-189. doi: 10.1016/j.jep.2017.12.018. Epub 2017 Dec 15.
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Nerve Growth Factor Protects Retinal Ganglion Cells Related to Inhibiting Endoplasmic Reticulum Stress by Inhibiting IRE1-JNK-CHOP Signaling Pathway.神经生长因子通过抑制 IRE1-JNK-CHOP 信号通路保护与内质网应激相关的视网膜神经节细胞。
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Role of endoplasmic reticulum stress in the loss of retinal ganglion cells in diabetic retinopathy.内质网应激在糖尿病性视网膜病变中视网膜神经节细胞丢失中的作用。
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Protective effect of Liraglutide on diabetic retinal neurodegeneration via inhibiting oxidative stress and endoplasmic reticulum stress.利拉鲁肽通过抑制氧化应激和内质网应激对糖尿病性视网膜神经退行性变的保护作用。
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Glucose-Regulated Protein 78, via Releasing β-Catenin from Adherens Junctions, Facilitates Its Interaction with STAT3 in Mediating Retinal Neovascularization.葡萄糖调节蛋白 78 通过从黏着连接释放 β-连环蛋白促进其与 STAT3 相互作用介导视网膜新生血管形成。
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Knockdown of MALAT1 attenuates high-glucose-induced angiogenesis and inflammation via endoplasmic reticulum stress in human retinal vascular endothelial cells.敲低 MALAT1 通过内质网应激减轻高糖诱导的人视网膜血管内皮细胞血管生成和炎症反应。
Biomed Pharmacother. 2020 Apr;124:109699. doi: 10.1016/j.biopha.2019.109699. Epub 2020 Jan 25.
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Role of endoplasmic reticulum stress in 12/15-lipoxygenase-induced retinal microvascular dysfunction in a mouse model of diabetic retinopathy.内质网应激在 12/15-脂氧合酶诱导的糖尿病视网膜病变小鼠模型视网膜微血管功能障碍中的作用。
Diabetologia. 2018 May;61(5):1220-1232. doi: 10.1007/s00125-018-4560-z. Epub 2018 Feb 21.

Author Correction: GLCCI1 alleviates GRP78-initiated endoplasmic reticulum stress-induced apoptosis of retinal ganglion cells in diabetic retinopathy by upregulating and interacting with HSP90AB1.

作者信息

Liu Jiayou, Yu Hongdan, Yu Shengxue, Liu Mengren, Chen Xinyuan, Wang Yufei, Li Jing, Shi Caixing, Liu Wenqiang, Zuo Zhongfu, Liu Xuezheng

机构信息

School of Basic Medical Sciences, Guangxi Medical University, Nanning City, People's Republic of China.

School of Basic and Forensic Medical Sciences, North Sichuan Medical College, Nanchong City, People's Republic of China.

出版信息

Sci Rep. 2024 Dec 16;14(1):30500. doi: 10.1038/s41598-024-83001-6.

DOI:10.1038/s41598-024-83001-6
PMID:39681632
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11649925/
Abstract
摘要