Dysregulated Proteostasis-induced Ferroptosis in Gastroesophageal Reflux Disease: Hero or Villain?
作者信息
Wang David H
机构信息
Division of Hematology and Oncology, University of Michigan, Ann Arbor, Michigan; Veterans Affairs Ann Arbor Healthcare System, Ann Arbor, Michigan.
出版信息
Cell Mol Gastroenterol Hepatol. 2025;19(3):101445. doi: 10.1016/j.jcmgh.2024.101445. Epub 2025 Jan 17.
Abstract
摘要
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Dysregulated Proteostasis-induced Ferroptosis in Gastroesophageal Reflux Disease: Hero or Villain?胃食管反流病中蛋白稳态失调诱导的铁死亡：英雄还是反派？
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本文引用的文献
1
Chronic Gastroesophageal Reflux Dysregulates Proteostasis in Esophageal Epithelial Cells.慢性胃食管反流会扰乱食管上皮细胞中的蛋白质稳态。
Cell Mol Gastroenterol Hepatol. 2025;19(3):101434. doi: 10.1016/j.jcmgh.2024.101434. Epub 2024 Dec 3.
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Protein adduction causes non-mutational inhibition of p53 tumor suppressor.蛋白质加合物导致 p53 肿瘤抑制因子非突变性抑制。
Cell Rep. 2023 Jan 31;42(1):112024. doi: 10.1016/j.celrep.2023.112024. Epub 2023 Jan 23.
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The role of ferroptosis in esophageal cancer.铁死亡在食管癌中的作用。
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Broadening horizons: the role of ferroptosis in cancer.拓宽视野：铁死亡在癌症中的作用。
Nat Rev Clin Oncol. 2021 May;18(5):280-296. doi: 10.1038/s41571-020-00462-0. Epub 2021 Jan 29.
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Gastroesophageal Reflux Induces Protein Adducts in the Esophagus.胃食管反流会在食管中诱导蛋白质加合物的形成。
Cell Mol Gastroenterol Hepatol. 2019;7(2):480-482.e7. doi: 10.1016/j.jcmgh.2018.10.017. Epub 2018 Nov 8.
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Bile acid and inflammation activate gastric cardia stem cells in a mouse model of Barrett-like metaplasia.胆汁酸和炎症激活了巴雷特样化生小鼠模型中的胃贲门干细胞。
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Inflammation and oxidative stress in gastroesophageal reflux disease.胃食管反流病中的炎症和氧化应激。
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