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O-连接的N-乙酰葡糖胺化决定细胞焦亡。

O-GlcNAcylation dictates pyroptosis.

作者信息

Lang Yue, Li Jincheng, Zhang Leiliang

机构信息

Department of Clinical Laboratory Medicine, The First Affiliated Hospital of Shandong First Medical University and Shandong Provincial Qianfoshan Hospital, Jinan, Shandong, China.

Department of Pathogen Biology, School of Clinical and Basic Medical Sciences, Shandong First Medical University and Shandong Academy of Medical Sciences, Jinan, Shandong, China.

出版信息

Front Immunol. 2024 Dec 17;15:1513542. doi: 10.3389/fimmu.2024.1513542. eCollection 2024.

Abstract

O-GlcNAcylation is a dynamic post-translational modification involving the attachment of N-acetylglucosamine to serine and threonine residues. This review emphasizes its role in regulating the signaling pathways of pyroptosis. Specifically, the O-GlcNAcylation of GSDMD is linked to the modulation of pyroptosis, suggesting that enhancing O-GlcNAcylation of GSDMD could be crucial for improving hypoperfusion in sepsis. Additionally, GSDME, another member of the gasdermin family, facilitates macrophage pyroptosis through O-GlcNAcylation induced by high glucose levels in the context of periodontitis. The review also examines the effects of O-GlcNAcylation on the NLRP3 inflammasome and its regulators, including NEK7 and NF-κB. Overall, this review emphasizes the role of O-GlcNAcylation in the pathogenesis of conditions such as sepsis, periodontitis, and osteoarthritis, identifying potential therapeutic targets for managing inflammatory responses through its targeted modulation.

摘要

O-连接的N-乙酰葡糖胺化是一种动态的翻译后修饰,涉及将N-乙酰葡糖胺连接到丝氨酸和苏氨酸残基上。本综述强调了其在调节细胞焦亡信号通路中的作用。具体而言,GSDMD的O-连接的N-乙酰葡糖胺化与细胞焦亡的调节有关,这表明增强GSDMD的O-连接的N-乙酰葡糖胺化对于改善脓毒症中的低灌注可能至关重要。此外,gasdermin家族的另一个成员GSDME在牙周炎的情况下通过高糖水平诱导的O-连接的N-乙酰葡糖胺化促进巨噬细胞焦亡。该综述还研究了O-连接的N-乙酰葡糖胺化对NLRP3炎性小体及其调节因子(包括NEK7和NF-κB)的影响。总体而言,本综述强调了O-连接的N-乙酰葡糖胺化在脓毒症、牙周炎和骨关节炎等病症发病机制中的作用,通过其靶向调节确定了管理炎症反应的潜在治疗靶点。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f48/11685218/3449dac1e18b/fimmu-15-1513542-g001.jpg

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