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Correction: Downregulation of miR-223 promotes HMGB2 expression and induces oxidative stress to activate JNK and promote autophagy in an in vitro model of acute lung injury.

作者信息

Tan Hao-Yu, Qing Bei, Luo Xian-Mei, Liang Heng-Xing

机构信息

Department of Cardio-Vascular Surgery, the Second Xiangya Hospital of Central South University, No.139 Middle Renmin Road, Hunan Province, 410011, Changsha, People's Republic of China.

Department of Thoracic Surgery, the Second Xiangya Hospital of Central South University, No.139 Middle Renmin Road, Hunan Province, 410011, Changsha, People's Republic of China.

出版信息

J Inflamm (Lond). 2025 Jan 6;22(1):1. doi: 10.1186/s12950-024-00424-8.

DOI:10.1186/s12950-024-00424-8
PMID:39762912
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11702141/
Abstract
摘要

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Correction: Downregulation of miR-223 promotes HMGB2 expression and induces oxidative stress to activate JNK and promote autophagy in an in vitro model of acute lung injury.更正：在急性肺损伤的体外模型中，miR-223的下调促进HMGB2表达并诱导氧化应激以激活JNK并促进自噬。
J Inflamm (Lond). 2025 Jan 6;22(1):1. doi: 10.1186/s12950-024-00424-8.
2
Downregulation of miR-223 promotes HMGB2 expression and induces oxidative stress to activate JNK and promote autophagy in an in vitro model of acute lung injury.在急性肺损伤体外模型中，miR-223的下调促进HMGB2表达并诱导氧化应激以激活JNK并促进自噬。
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本文引用的文献

1
Downregulation of miR-223 promotes HMGB2 expression and induces oxidative stress to activate JNK and promote autophagy in an in vitro model of acute lung injury.在急性肺损伤体外模型中，miR-223的下调促进HMGB2表达并诱导氧化应激以激活JNK并促进自噬。
J Inflamm (Lond). 2021 Nov 3;18(1):29. doi: 10.1186/s12950-021-00295-3.