卡托普利对高血压患者肾素 - 血管紧张素系统的阻断可导致醛固酮分泌持续减少、钾潴留和利钠作用。 - Suppr

Suppr

超能文献

Interruption of the renin-angiotensin system in hypertensive patients by captopril induces sustained reduction in aldosterone secretion, potassium retention and natruiresis.

作者信息

Atlas S A, Case D B, Sealey J E, Laragh J H, McKinstry D N

出版信息

Hypertension. 1979 May-Jun;1(3):274-80. doi: 10.1161/01.hyp.1.3.274.

DOI:10.1161/01.hyp.1.3.274

PMID:399239

Abstract

摘要

相似文献

Interruption of the renin-angiotensin system in hypertensive patients by captopril induces sustained reduction in aldosterone secretion, potassium retention and natruiresis.卡托普利对高血压患者肾素 - 血管紧张素系统的阻断可导致醛固酮分泌持续减少、钾潴留和利钠作用。

Hypertension. 1979 May-Jun;1(3):274-80. doi: 10.1161/01.hyp.1.3.274.

Captopril in clinical hypertension. Changes in components of renin-angiotensin system and in body composition in relation to fall in blood pressure with a note on measurement of angiotensin II during converting enzyme inhibition.卡托普利治疗临床高血压。肾素 - 血管紧张素系统各组分及身体成分的变化与血压下降的关系，并附关于转换酶抑制期间血管紧张素II测量的说明

Br Heart J. 1980 Sep;44(3):290-6. doi: 10.1136/hrt.44.3.290.

Renal artery stenosis with normal angiotensin II values. Relationship between angiotensin II and body sodium and potassium on correction of hypertension by captopril and subsequent surgery.肾动脉狭窄伴血管紧张素II值正常。血管紧张素II与机体钠和钾之间的关系：关于卡托普利纠正高血压及后续手术的研究

Hypertension. 1981 Jan-Feb;3(1):53-8. doi: 10.1161/01.hyp.3.1.53.

Captopril in hypertension with renal artery stenosis and in intractable hypertension; acute and chronic changes in circulating concentrations of renin, angiotensins I and II and aldosterone, and in body composition.卡托普利治疗肾动脉狭窄性高血压和顽固性高血压；肾素、血管紧张素I和II以及醛固酮循环浓度的急性和慢性变化，以及身体成分的变化。

Clin Sci (Lond). 1979 Dec;57 Suppl 5:139s-143s. doi: 10.1042/cs057139s.

The renin--angiotensin--aldosterone system in the maintenance of blood pressure, aldosterone secretion and sodium balance in normotensive subjects.肾素-血管紧张素-醛固酮系统在维持正常血压者的血压、醛固酮分泌及钠平衡中的作用。

Clin Sci (Lond). 1980 Dec;59 Suppl 6:95s-99s. doi: 10.1042/cs059095s.

Maintenance of blood pressure by the renin-angiotensin system in normal man.

Nature. 1981 May 28;291(5813):329-31. doi: 10.1038/291329a0.

Hyponatraemic hypertensive syndrome with renal-artery occlusion corrected by captopril.卡托普利纠正的伴有肾动脉闭塞的低钠血症性高血压综合征。

Lancet. 1979 Sep 22;2(8143):606-9. doi: 10.1016/s0140-6736(79)91666-0.

Effect of angiotensin converting enzyme inhibitor on serum aldosterone and potassium level.血管紧张素转换酶抑制剂对血清醛固酮和钾水平的影响。

Jpn Heart J. 1982 Mar;23(2):191-9. doi: 10.1536/ihj.23.191.

[The pressor and adrenal responses to angiotensin II in patients with normoreninemic essential hypertension--the influence of sodium intake and captopril].

Nihon Jinzo Gakkai Shi. 1983 Feb;25(2):179-88.

[An experimental study on the effects of the chronic administration of an angiotensin converting enzyme inhibitor (captopril) on blood pressure and the renin-angiotensin-aldosterone system (author's transl)].血管紧张素转换酶抑制剂（卡托普利）长期给药对血压及肾素 - 血管紧张素 - 醛固酮系统影响的实验研究（作者译）

Nihon Naibunpi Gakkai Zasshi. 1980 Jun 20;56(6):868-83. doi: 10.1507/endocrine1927.56.6_868.

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Drug Saf. 1996 Sep;15(3):200-11. doi: 10.2165/00002018-199615030-00005.

Current drug treatment and treatment patterns with antihypertensive drugs.当前的药物治疗以及抗高血压药物的治疗模式。

Drugs. 1996 Jul;52(1):1-16. doi: 10.2165/00003495-199652010-00001.

OACE inhibition does not interfere with acute extrarenal or renal potassium disposal in chronic renal failure.

Eur J Clin Pharmacol. 1994;46(3):185-9. doi: 10.1007/BF00192546.

Pharmacokinetics and pharmacodynamics of ramipril and piretanide administered alone and in combination.单独及联合使用雷米普利和吡咯他尼的药代动力学和药效学。

Eur J Clin Pharmacol. 1994;46(6):545-50. doi: 10.1007/BF00196113.

Captopril treatment: inter-dose variations in renin, angiotensins I and II, aldosterone and blood pressure.卡托普利治疗：肾素、血管紧张素I和II、醛固酮及血压的剂间变化

Br J Clin Pharmacol. 1982 Jun;13(6):855-8. doi: 10.1111/j.1365-2125.1982.tb01878.x.

Effect of captopril on renal function in patients with congestive heart failure.卡托普利对充血性心力衰竭患者肾功能的影响。

Br Heart J. 1981 Nov;46(5):522-7. doi: 10.1136/hrt.46.5.522.

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