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Induction of micronuclei by benzene in B6C3F1 mice: retrospective analysis of peripheral blood smears from the NTP carcinogenesis bioassay.

作者信息

Choy W N, MacGregor J T, Shelby M D, Maronpot R R

出版信息

Mutat Res. 1985 May-Jun;143(1-2):55-9. doi: 10.1016/0165-7992(85)90105-8.

DOI:10.1016/0165-7992(85)90105-8
PMID:4000143
Abstract
摘要

相似文献

1
Induction of micronuclei by benzene in B6C3F1 mice: retrospective analysis of peripheral blood smears from the NTP carcinogenesis bioassay.
Mutat Res. 1985 May-Jun;143(1-2):55-9. doi: 10.1016/0165-7992(85)90105-8.
2
Micronuclei induced in peripheral blood of E mu-PIM-1 transgenic mice by chronic oral treatment with 2-acetylaminofluorene or benzene but not with diethyl-nitrosamine or 1,2-dichloroethane.通过长期口服2-乙酰氨基芴或苯而非二乙基亚硝胺或1,2-二氯乙烷诱导Eμ-PIM-1转基因小鼠外周血中的微核。
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The production of micronuclei from chromosome aberrations by chemical carcinogens in mice.
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[Analysis of normochromic erythrocytes with micronuclei in the peripheral blood of rats after peroral administration of benzol].[经口给予苯后大鼠外周血中含微核的正色素红细胞分析]
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The effect of exposure regimen and duration on benzene-induced bone marrow damage in mice. II. Strain comparisons involving B6C3F1, C57B1/6 and DBA/2 male mice.暴露方案和持续时间对苯诱导的小鼠骨髓损伤的影响。II. 涉及B6C3F1、C57B1/6和DBA/2雄性小鼠的品系比较。
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NTP Toxicology and carcinogenesis studies of bromodichloromethane (CAS No. 75-27-4) in male F344/N rats and female B6C3F1 mice (Drinking Water Studies).NTP对雄性F344/N大鼠和雌性B6C3F1小鼠进行的溴二氯甲烷（CAS编号：75-27-4）毒理学和致癌性研究（饮用水研究）
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[The micronucleus test: characteristics of the micronuclei count and the method of statistical data processing].[微核试验：微核计数特征及统计数据处理方法]
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Induction of micronuclei and nuclear abnormalities in the erythrocytes of mudminnows (Umbra limi) and brown bullheads (Ictalurus nebulosus).诱导北美泥鱼（Umbra limi）和棕牛头鱼（Ictalurus nebulosus）红细胞中的微核和核异常。
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10
Cytologic and cytogenetic effects of benzene.
J Toxicol Environ Health Suppl. 1977;2:63-8.

引用本文的文献

1
Mechanistic considerations in benzene physiological model development.苯生理模型开发中的机制考量
Environ Health Perspect. 1996 Dec;104 Suppl 6(Suppl 6):1399-404. doi: 10.1289/ehp.961041399.
2
Possible implications from results of animal studies in human risk estimations for benzene: nonlinear dose-response relationship due to saturation of metabolism.动物研究结果对苯的人类风险评估的潜在影响：由于代谢饱和导致的非线性剂量反应关系。
J Cancer Res Clin Oncol. 1987;113(4):349-58. doi: 10.1007/BF00397718.
3
Effect of exposure route, regimen, and duration on benzene-induced genotoxic and cytotoxic bone marrow damage in mice.
暴露途径、给药方案和持续时间对苯诱导的小鼠骨髓遗传毒性和细胞毒性损伤的影响。
Environ Health Perspect. 1989 Jul;82:65-74. doi: 10.1289/ehp.898265.
4
Prevention of benzene-induced myelotoxicity by nonsteroidal anti-inflammatory drugs.非甾体抗炎药预防苯诱导的骨髓毒性
Environ Health Perspect. 1989 Jul;82:57-64. doi: 10.1289/ehp.898257.
5
Results of animal studies suggest a nonlinear dose-response relationship for benzene effects.动物研究结果表明，苯效应存在非线性剂量反应关系。
Environ Health Perspect. 1989 Jul;82:171-6. doi: 10.1289/ehp.8982171.
6
Multiple-site carcinogenicity of benzene in Fischer 344 rats and B6C3F1 mice.苯在Fischer 344大鼠和B6C3F1小鼠中的多部位致癌性。
Environ Health Perspect. 1989 Jul;82:125-63. doi: 10.1289/ehp.8982125.
7
Peroxidase-dependent metabolism of benzene's phenolic metabolites and its potential role in benzene toxicity and carcinogenicity.苯酚类代谢产物的过氧化物酶依赖性代谢及其在苯毒性和致癌性中的潜在作用。
Environ Health Perspect. 1989 Jul;82:23-9. doi: 10.1289/ehp.898223.