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因栓子阻塞及脑动脉立即再通导致的短暂性神经功能缺损。

Transient neurological deficits due to embolic occlusion and immediate reopening of the cerebral arteries.

作者信息

Taneda M, Shimada N, Tsuchiya T

出版信息

Stroke. 1985 May-Jun;16(3):522-4. doi: 10.1161/01.str.16.3.522.

Abstract

The authors present two cases of transient occlusion of the major cerebral arteries which occurred during transfemoral catheterization of the carotid artery. Right hemiplegia and aphasia developed suddenly in both cases, and disappeared completely within 14 hours in one case and 25 minutes in the other. On the angiograms performed at the moment of onset of the symptoms, the site of the occlusion was the left internal carotid artery in one case and the left middle cerebral artery in the other. Angiograms which were repeated soon after clinical improvement revealed complete dissolution of the occluding emboli. These cases present direct radiographic evidence that embolic occlusion of a major cerebral artery and its disappearance is the mechanism of the transient manifestation of the neurological deficits associated with cerebrovascular catheterization.

摘要

作者报告了两例在经股动脉行颈动脉导管插入术期间发生的大脑主要动脉短暂闭塞病例。两例均突然出现右侧偏瘫和失语,其中一例在14小时内完全消失,另一例在25分钟内完全消失。症状发作时所做的血管造影显示,一例闭塞部位为左颈内动脉,另一例为左大脑中动脉。临床症状改善后不久重复进行的血管造影显示,闭塞性栓子完全溶解。这些病例提供了直接的影像学证据,表明大脑主要动脉的栓塞性闭塞及其消失是与脑血管导管插入术相关的神经功能缺损短暂表现的机制。

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