The importance of disrupting complement activation in acute lung injury.
作者信息
Kapur Rick, Semple John W, Vlaar Alexander Pj
机构信息
Sanquin Research, Department of Experimental Immunohematology, Amsterdam, Netherlands.
Landsteiner Laboratory, Amsterdam University Medical Center, University of Amsterdam, Amsterdam, Netherlands.
出版信息
J Clin Invest. 2024 Jun 17;134(12):e182251. doi: 10.1172/JCI182251.
Abstract
摘要
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本文引用的文献
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Complement activation drives antibody-mediated transfusion-related acute lung injury via macrophage trafficking and formation of NETs.补体激活通过巨噬细胞转运和中性粒细胞胞外陷阱的形成驱动抗体介导的输血相关急性肺损伤。
Blood. 2024 Jan 4;143(1):79-91. doi: 10.1182/blood.2023020484.
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Mechanism and intervention of murine transfusion-related acute lung injury caused by anti-CD36 antibodies.抗 CD36 抗体引起的鼠输血相关性急性肺损伤的机制及干预。
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Anti-C5a antibody (vilobelimab) therapy for critically ill, invasively mechanically ventilated patients with COVID-19 (PANAMO): a multicentre, double-blind, randomised, placebo-controlled, phase 3 trial.抗 C5a 抗体（vilobelimab）治疗 COVID-19 危重症、有创机械通气患者（PANAMO）：一项多中心、双盲、随机、安慰剂对照、3 期临床试验。
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Biological and structural characterization of murine TRALI antibody reveals increased Fc-mediated complement activation.小鼠输血相关急性肺损伤抗体的生物学和结构特征揭示Fc介导的补体激活增加。
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