Koranda F C, Webster R C
Arch Otolaryngol. 1985 Jul;111(7):421-4. doi: 10.1001/archotol.1985.00800090035002.
The trapdoor effect is an elevated and bulging deformity of tissue within the semicircular confines of a U-, C-, or V-shaped scar. Various theories to explain this phenomenon are lymphatic and venous obstruction, hypertrophy of the scar, excessive fatty and redundant tissue, beveled wound edges, and contracture of the scar. Our data suggest that scar contracture is the predominant cause of the trapdoor effect in nasolabial flaps. For mild to moderately severe trapdoor deformities, multiple, small Z-plasties about the periphery of the nasolabial flap are indicated. Intralesional triamcinolone acetonide injections may produce a "pharmacologic Z-plasty" effect in some trapdoor deformities. For marked trapdoor deformities, the combination of multiple, small Z-plasties along the semicircular scar and peripheral undermining about the trapdoor defect is the corrective procedure. The trapdoor deformity may be prevented or lessened by peripheral undermining about the recipient site of the flap equal to or greater in area than the recipient site.
活板门效应是指在U形、C形或V形瘢痕的半圆形范围内组织出现隆起和鼓出的畸形。关于这一现象的各种理论包括淋巴和静脉阻塞、瘢痕肥大、过多的脂肪和冗余组织、伤口边缘呈斜面以及瘢痕挛缩。我们的数据表明,瘢痕挛缩是鼻唇瓣活板门效应的主要原因。对于轻度至中度严重的活板门畸形,建议在鼻唇瓣周边进行多个小Z成形术。病灶内注射曲安奈德在一些活板门畸形中可能产生“药物性Z成形术”效果。对于明显的活板门畸形,沿半圆形瘢痕进行多个小Z成形术并对活板门缺损周边进行皮下分离是矫正方法。通过在皮瓣受区周边进行面积等于或大于受区的皮下分离,可预防或减轻活板门畸形。