Shukla G S
Environ Res. 1985 Aug;37(2):349-54. doi: 10.1016/0013-9351(85)90115-x.
Exposure of freshwater prawn (Macrobrachium lamarrei (H. Milne Edwards) to a lethal dose (96-hr LC50;0.78 mg/l) of dichlorvos for 24, 48, 72, and 96 hr induced inhibition in acetylcholinesterase and alkaline phosphatase activities and enhanced acid phosphatase activity. Hepatic glycogen values were depleted while the blood glucose level was increased up to 72 hr of exposure. After 96 hr of dichlorvos exposure hypoglycemia was recorded. The possible role of these changes in the dichlorvos intoxication is discussed.
将淡水虾（罗氏沼虾（H. 米尔恩·爱德华兹））暴露于致死剂量（96小时半数致死浓度；0.78毫克/升）的敌敌畏中24、48、72和96小时，会导致乙酰胆碱酯酶和碱性磷酸酶活性受到抑制，酸性磷酸酶活性增强。肝糖原值减少，而血糖水平在暴露72小时内升高。在敌敌畏暴露96小时后，出现低血糖。讨论了这些变化在敌敌畏中毒中的可能作用。
