Correction to "Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing".
出版信息
Wound Repair Regen. 2025 Mar-Apr;33(2):e70024. doi: 10.1111/wrr.70024.
Abstract
摘要
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1
Correction to "Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing".对“TRIM72介导的内质网应激抑制促进FOXM1对糖尿病溃疡愈合的促进作用”的修正
Wound Repair Regen. 2025 Mar-Apr;33(2):e70024. doi: 10.1111/wrr.70024.
2
Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing.抑制TRIM72介导的内质网应激促进FOXM1对糖尿病溃疡愈合的促进作用。
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KDELR3 is transcriptionally activated by FOXM1 and accelerates lung adenocarcinoma growth and metastasis via inhibiting endoplasmic reticulum stress-induced cell apoptosis.KDELR3由FOXM1转录激活，并通过抑制内质网应激诱导的细胞凋亡来加速肺腺癌的生长和转移。
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Role of the FOXM1/CMA/ER stress axis in regulating the progression of nonalcoholic steatohepatitis.FOXM1/CMA/内质网应激轴在调控非酒精性脂肪性肝炎进展中的作用
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Regulation of FOXM1 by HDAC3 Inhibition Ameliorates Macrophage Endoplasmic Reticulum stress and Apoptosis in Mycobacterium tuberculosis Infection.通过抑制HDAC3对FOXM1进行调控可改善结核分枝杆菌感染中巨噬细胞的内质网应激和凋亡。
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FOXM1 network in association with TREM1 suppression regulates NET formation in diabetic foot ulcers.FOXM1 网络与 TREM1 抑制共同调节糖尿病足溃疡中的 NET 形成。
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Vitamin D Ameliorates Impaired Wound Healing in Streptozotocin-Induced Diabetic Mice by Suppressing Endoplasmic Reticulum Stress.维生素 D 通过抑制内质网应激改善链脲佐菌素诱导的糖尿病小鼠的伤口愈合受损。
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