• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Correction to "Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing".

出版信息

Wound Repair Regen. 2025 Mar-Apr;33(2):e70024. doi: 10.1111/wrr.70024.

DOI:10.1111/wrr.70024
PMID:40211807
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12128704/
Abstract
摘要

相似文献

1
Correction to "Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing".对“TRIM72介导的内质网应激抑制促进FOXM1对糖尿病溃疡愈合的促进作用”的修正
Wound Repair Regen. 2025 Mar-Apr;33(2):e70024. doi: 10.1111/wrr.70024.
2
Suppression of TRIM72-mediated endoplasmic reticulum stress facilitates FOXM1 promotion of diabetic ulcer healing.抑制TRIM72介导的内质网应激促进FOXM1对糖尿病溃疡愈合的促进作用。
Wound Repair Regen. 2025 Jan-Feb;33(1):e13247. doi: 10.1111/wrr.13247.
3
Identification of signalling downstream of the transcription factor forkhead box protein M1 that protects against endoplasmic reticulum stress in a diabetic foot ulcer model.鉴定转录因子叉头框蛋白 M1 下游的信号通路，该通路在糖尿病足溃疡模型中具有抵抗内质网应激的作用。
Diabet Med. 2023 Jun;40(6):e15051. doi: 10.1111/dme.15051. Epub 2023 Feb 15.
4
FOXM1 accelerates wound healing in diabetic foot ulcer by inducing M2 macrophage polarization through a mechanism involving SEMA3C/NRP2/Hedgehog signaling.FOXM1 通过 SEMA3C/NRP2/Hedgehog 信号通路诱导 M2 巨噬细胞极化加速糖尿病足溃疡愈合。
Diabetes Res Clin Pract. 2022 Feb;184:109121. doi: 10.1016/j.diabres.2021.109121. Epub 2021 Nov 3.
5
KDELR3 is transcriptionally activated by FOXM1 and accelerates lung adenocarcinoma growth and metastasis via inhibiting endoplasmic reticulum stress-induced cell apoptosis.KDELR3由FOXM1转录激活，并通过抑制内质网应激诱导的细胞凋亡来加速肺腺癌的生长和转移。
Hum Cell. 2025 May 24;38(4):106. doi: 10.1007/s13577-025-01238-3.
6
Role of the FOXM1/CMA/ER stress axis in regulating the progression of nonalcoholic steatohepatitis.FOXM1/CMA/内质网应激轴在调控非酒精性脂肪性肝炎进展中的作用
Clin Transl Med. 2025 Feb;15(2):e70202. doi: 10.1002/ctm2.70202.
7
Regulation of FOXM1 by HDAC3 Inhibition Ameliorates Macrophage Endoplasmic Reticulum stress and Apoptosis in Mycobacterium tuberculosis Infection.通过抑制HDAC3对FOXM1进行调控可改善结核分枝杆菌感染中巨噬细胞的内质网应激和凋亡。
Immunobiology. 2025 Mar;230(2):152879. doi: 10.1016/j.imbio.2025.152879. Epub 2025 Feb 5.
8
FOXM1 network in association with TREM1 suppression regulates NET formation in diabetic foot ulcers.FOXM1 网络与 TREM1 抑制共同调节糖尿病足溃疡中的 NET 形成。
EMBO Rep. 2022 Aug 3;23(8):e54558. doi: 10.15252/embr.202154558. Epub 2022 Jul 20.
9
Vitamin D Ameliorates Impaired Wound Healing in Streptozotocin-Induced Diabetic Mice by Suppressing Endoplasmic Reticulum Stress.维生素 D 通过抑制内质网应激改善链脲佐菌素诱导的糖尿病小鼠的伤口愈合受损。
J Diabetes Res. 2018 Mar 7;2018:1757925. doi: 10.1155/2018/1757925. eCollection 2018.
10
Exploring the mechanism by which UCHL3 alleviates diabetic foot ulcers: FOXM1/NLRP3 inflammasome-mediated angiogenesis and endothelial cell pyroptosis.探索UCHL3减轻糖尿病足溃疡的机制：FOXM1/NLRP3炎性小体介导的血管生成和内皮细胞焦亡
J Orthop Surg Res. 2025 May 20;20(1):488. doi: 10.1186/s13018-025-05914-w.