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Fatty acid profiles in aldosterone-producing adenoma: insights into pathogenetic significance.

作者信息

Murakami Masanori

机构信息

Department of Molecular Endocrinology and Metabolism, Graduate School of Medical and Dental Sciences, Institute of Science Tokyo, Tokyo, 113-8510, Japan.

出版信息

Hypertens Res. 2025 Apr 16. doi: 10.1038/s41440-025-02211-1.

DOI:10.1038/s41440-025-02211-1
PMID:40240872
Abstract
摘要

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Hypertens Res. 2025 Apr 16. doi: 10.1038/s41440-025-02211-1.
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3
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Different pathogenesis of glucose intolerance in two subtypes of primary aldosteronism: Aldosterone-producing adenoma and idiopathic hyperaldosteronism.两种原发性醛固酮增多症亚型（醛固酮瘤和特发性醛固酮增多症）中葡萄糖不耐受的不同发病机制。
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[The genetic background of primary aldosteronism].[原发性醛固酮增多症的遗传背景]
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Imaging of aldosterone-secreting adenomas: a prospective comparison of computed tomography and magnetic resonance imaging in 27 patients with suspected primary aldosteronism.醛固酮分泌性腺瘤的影像学检查：27例疑似原发性醛固酮增多症患者的计算机断层扫描与磁共振成像前瞻性比较
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本文引用的文献

1
Primary Aldosteronism: Spatial Multiomics Mapping of Genotype-Dependent Heterogeneity and Tumor Expansion of Aldosterone-Producing Adenomas.原发性醛固酮增多症：醛固酮瘤基因型依赖性异质性和肿瘤扩张的空间多组学图谱。
Hypertension. 2023 Jul;80(7):1555-1567. doi: 10.1161/HYPERTENSIONAHA.123.20921. Epub 2023 May 1.
2
In Situ Metabolomics of Cortisol-Producing Adenomas.皮质醇分泌腺瘤的原位代谢组学。
Clin Chem. 2023 Feb 1;69(2):149-159. doi: 10.1093/clinchem/hvac191.
3
Phenotype-genotype correlation in aldosterone-producing adenomas characterized by intracellular cholesterol metabolism.
醛固酮腺瘤细胞内胆固醇代谢特征的表型-基因型相关性。
J Steroid Biochem Mol Biol. 2022 Jul;221:106116. doi: 10.1016/j.jsbmb.2022.106116. Epub 2022 May 6.
4
International Histopathology Consensus for Unilateral Primary Aldosteronism.国际单侧原发性醛固酮增多症组织病理学共识。
J Clin Endocrinol Metab. 2021 Jan 1;106(1):42-54. doi: 10.1210/clinem/dgaa484.
5
Genetic and Genomic Mechanisms of Primary Aldosteronism.原发性醛固酮增多症的遗传和基因组机制。
Trends Mol Med. 2020 Sep;26(9):819-832. doi: 10.1016/j.molmed.2020.05.005. Epub 2020 Jun 18.
6
Tissue-Specific Ablation of ACSL4 Results in Disturbed Steroidogenesis.组织特异性敲除 ACSL4 导致类固醇生成紊乱。
Endocrinology. 2019 Nov 1;160(11):2517-2528. doi: 10.1210/en.2019-00464.
7
In situ metabolomics of aldosterone-producing adenomas.醛固酮瘤的原位代谢组学。
JCI Insight. 2019 Sep 5;4(17):130356. doi: 10.1172/jci.insight.130356.
8
Tumor Cell Subtypes Based on the Intracellular Hormonal Activity in KCNJ5-Mutated Aldosterone-Producing Adenoma.基于 KCNJ5 突变型醛固酮瘤细胞内激素活性的肿瘤细胞亚型。
Hypertension. 2018 Sep;72(3):632-640. doi: 10.1161/HYPERTENSIONAHA.118.10907.
9
Molecular characteristics of the KCNJ5 mutated aldosterone-producing adenomas.醛固酮瘤中 KCNJ5 突变的分子特征。
Endocr Relat Cancer. 2017 Oct;24(10):531-541. doi: 10.1530/ERC-17-0117. Epub 2017 Jul 26.