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Protecting the brain from multifaceted damage and promoting recovery.保护大脑免受多方面损伤并促进恢复。
Neuroprotection. 2025 Mar;3(1):1-4. doi: 10.1002/nep3.70003. Epub 2025 Mar 31.
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Post-ischemic protein restriction induces sustained neuroprotection, neurological recovery, brain remodeling, and gut microbiota rebalancing.缺血后蛋白质限制可诱导持续的神经保护、神经功能恢复、大脑重塑和肠道微生物组再平衡。
Brain Behav Immun. 2022 Feb;100:134-144. doi: 10.1016/j.bbi.2021.11.016. Epub 2021 Nov 27.
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Selenium supplementation provides potent neuroprotection following cerebral ischemia in mice.补硒可提供强烈的神经保护作用，减少脑缺血后的损伤。
J Cereb Blood Flow Metab. 2023 Jul;43(7):1060-1076. doi: 10.1177/0271678X231156981. Epub 2023 Feb 9.
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Mailuoning oral liquid attenuates convalescent cerebral ischemia by inhibiting AMPK/mTOR-associated apoptosis and promoting CREB/BDNF-mediated neuroprotection.脉络宁口服液通过抑制 AMPK/mTOR 相关凋亡和促进 CREB/BDNF 介导的神经保护作用减轻恢复期脑缺血。
J Ethnopharmacol. 2023 Dec 5;317:116731. doi: 10.1016/j.jep.2023.116731. Epub 2023 Jun 3.
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Mechanisms of time-restricted feeding-induced neuroprotection and neuronal plasticity in ischemic stroke as a function of circadian rhythm.限时进食诱导的缺血性中风神经保护和神经元可塑性的机制是昼夜节律的功能。
Exp Neurol. 2025 Jan;383:115045. doi: 10.1016/j.expneurol.2024.115045. Epub 2024 Nov 6.
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Microglia as the Critical Regulators of Neuroprotection and Functional Recovery in Cerebral Ischemia.小胶质细胞作为脑缺血中神经保护和功能恢复的关键调节者。
Cell Mol Neurobiol. 2022 Nov;42(8):2505-2525. doi: 10.1007/s10571-021-01145-9. Epub 2021 Aug 30.
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N2 neutrophils may participate in spontaneous recovery after transient cerebral ischemia by inhibiting ischemic neuron injury in rats.N2 中性粒细胞可能通过抑制大鼠短暂性脑缺血后的缺血性神经元损伤，参与自发性恢复。
Int Immunopharmacol. 2019 Dec;77:105970. doi: 10.1016/j.intimp.2019.105970. Epub 2019 Oct 29.
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Cognitive Deficits after Cerebral Ischemia and Underlying Dysfunctional Plasticity: Potential Targets for Recovery of Cognition.脑缺血后的认知缺陷及潜在的功能失调可塑性：认知恢复的潜在靶点
J Alzheimers Dis. 2017;60(s1):S87-S105. doi: 10.3233/JAD-170057.
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Hypocaloric Diet Initiated Post-Ischemia Provides Long-Term Neuroprotection and Promotes Peri-Infarct Brain Remodeling by Regulating Metabolic and Survival-Promoting Proteins.缺血后低热量饮食可通过调节代谢和促进存活蛋白提供长期神经保护并促进梗死周围脑重塑。
Mol Neurobiol. 2021 Apr;58(4):1491-1503. doi: 10.1007/s12035-020-02207-7. Epub 2020 Nov 17.
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An Enriched Environment Promotes Motor Function through Neuroprotection after Cerebral Ischemia.丰富环境通过神经保护促进脑缺血后运动功能恢复。
Biomed Res Int. 2023 Feb 8;2023:4143633. doi: 10.1155/2023/4143633. eCollection 2023.
引用本文的文献
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Neuroglia and immune cells play different roles in neuroinflammation and neuroimmune response in post-stroke neural injury and repair.神经胶质细胞和免疫细胞在中风后神经损伤与修复过程中的神经炎症和神经免疫反应中发挥着不同作用。
Acta Pharmacol Sin. 2025 Aug 12. doi: 10.1038/s41401-025-01640-5.
本文引用的文献
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Gut microbiota in Alzheimer's disease: Understanding molecular pathways and potential therapeutic perspectives.阿尔茨海默病中的肠道微生物群：理解分子途径和潜在治疗前景。
Ageing Res Rev. 2025 Feb;104:102659. doi: 10.1016/j.arr.2025.102659. Epub 2025 Jan 10.
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Preserving cognitive function in patients with Alzheimer's disease: The Alzheimer's disease neuroprotection research initiative (ADNRI).在阿尔茨海默病患者中保留认知功能：阿尔茨海默病神经保护研究倡议（ADNRI）。
Neuroprotection. 2023 Dec;1(2):84-98. doi: 10.1002/nep3.23. Epub 2023 Sep 21.
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Most Promising Approaches to Improve Stroke Outcomes: The Stroke Treatment Academic Industry Roundtable XII Workshop.改善脑卒中预后的最有前景的方法：脑卒中治疗学术产业圆桌会议第十二次研讨会。
Stroke. 2023 Dec;54(12):3202-3213. doi: 10.1161/STROKEAHA.123.044279. Epub 2023 Oct 27.
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Microbiome and Metabolome Insights into the Role of the Gastrointestinal-Brain Axis in Parkinson's and Alzheimer's Disease: Unveiling Potential Therapeutic Targets.微生物组和代谢组学对帕金森病和阿尔茨海默病中胃肠-脑轴作用的见解：揭示潜在治疗靶点
Metabolites. 2022 Dec 5;12(12):1222. doi: 10.3390/metabo12121222.
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Aging Microbiota-Gut-Brain Axis in Stroke Risk and Outcome.衰老的微生物群-肠-脑轴与中风风险和结局。
Circ Res. 2022 Apr 15;130(8):1112-1144. doi: 10.1161/CIRCRESAHA.122.319983. Epub 2022 Apr 14.
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Arterial Hypertension Aggravates Innate Immune Responses after Experimental Stroke.动脉高血压加重实验性中风后的固有免疫反应。
Front Cell Neurosci. 2015 Nov 27;9:461. doi: 10.3389/fncel.2015.00461. eCollection 2015.
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The molecular chaperone Hsp90 modulates intermediate steps of amyloid assembly of the Parkinson-related protein alpha-synuclein.分子伴侣Hsp90调节帕金森相关蛋白α-突触核蛋白淀粉样蛋白组装的中间步骤。
J Biol Chem. 2009 Nov 6;284(45):31190-9. doi: 10.1074/jbc.M109.057240. Epub 2009 Sep 15.
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