• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Correction: S-propargyl-cysteine attenuates temporomandibular joint osteoarthritis by regulating macrophage polarization via Inhibition of JAK/STAT signaling.

作者信息

Cai Wenyi, Wu Antong, Lin Zhongxiao, Cao Wei, Pathak Janak L, Jaspers Richard T, Li Rui, Li Xin, Zheng Kaihan, Lin Yufu, Zhou Na, Zhang Xin, Zhu Yizhun, Zhang Qingbin

机构信息

Department of Temporomandibular Joint, School and Hospital of Stomatology, Guangdong Engineering Research Center of Oral Restoration and Reconstruction, Guangzhou Key Laboratory of Basic and Applied Research of Oral Regenerative Medicine, Guangzhou Medical University, 195 Dongfeng Road (West), Yuexiu District, Guangzhou, Guangdong, 510140, China.

Laboratory for Myology, Department of Human Movement Sciences, Faculty of Behavioural and Movement Sciences, Vrije Universiteit Amsterdam, Amsterdam Movement Science, Amsterdam, The Netherlands.

出版信息

Mol Med. 2025 Apr 28;31(1):156. doi: 10.1186/s10020-025-01214-5.

DOI:10.1186/s10020-025-01214-5
PMID:40295904
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12039147/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3f7a/12039147/0463be163317/10020_2025_1214_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3f7a/12039147/2ce5ff6627b8/10020_2025_1214_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3f7a/12039147/0463be163317/10020_2025_1214_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3f7a/12039147/2ce5ff6627b8/10020_2025_1214_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/3f7a/12039147/0463be163317/10020_2025_1214_Fig1_HTML.jpg

相似文献

1
Correction: S-propargyl-cysteine attenuates temporomandibular joint osteoarthritis by regulating macrophage polarization via Inhibition of JAK/STAT signaling.更正:S-炔丙基半胱氨酸通过抑制JAK/STAT信号通路调节巨噬细胞极化来减轻颞下颌关节骨关节炎。
Mol Med. 2025 Apr 28;31(1):156. doi: 10.1186/s10020-025-01214-5.
2
S-propargyl-cysteine attenuates temporomandibular joint osteoarthritis by regulating macrophage polarization via Inhibition of JAK/STAT signaling.S-炔丙基半胱氨酸通过抑制JAK/STAT信号通路调节巨噬细胞极化来减轻颞下颌关节骨关节炎。
Mol Med. 2025 Apr 7;31(1):128. doi: 10.1186/s10020-025-01186-6.
3
Mechanical stimulation improves rotator cuff tendon-bone healing via activating IL-4/JAK/STAT signaling pathway mediated macrophage M2 polarization.机械刺激通过激活IL-4/JAK/STAT信号通路介导的巨噬细胞M2极化来改善肩袖肌腱-骨愈合。
J Orthop Translat. 2022 Oct 6;37:78-88. doi: 10.1016/j.jot.2022.08.008. eCollection 2022 Nov.
4
Cell volume regulation modulates macrophage-related inflammatory responses via JAK/STAT signaling pathways.细胞体积调节通过 JAK/STAT 信号通路调节巨噬细胞相关的炎症反应。
Acta Biomater. 2024 Sep 15;186:286-299. doi: 10.1016/j.actbio.2024.07.046. Epub 2024 Aug 3.
5
Suberosin attenuates rheumatoid arthritis by repolarizing macrophages and inhibiting synovitis via the JAK/STAT signaling pathway.木栓质通过使巨噬细胞重新极化并经由JAK/STAT信号通路抑制滑膜炎来减轻类风湿性关节炎。
Arthritis Res Ther. 2025 Jan 21;27(1):12. doi: 10.1186/s13075-025-03481-3.
6
Mechanism of LMNB1 activating GPR84 through JAK-STAT pathway to mediate M2 macrophage polarization in lung cancer.LMNB1通过JAK-STAT通路激活GPR84介导肺癌中M2巨噬细胞极化的机制。
Hum Immunol. 2024 Nov;85(6):111150. doi: 10.1016/j.humimm.2024.111150. Epub 2024 Oct 2.
7
ERK Modulates Macrophage Polarization and Alters Exosome miRNA Expression in Diabetic Nephropathy.ERK 调节巨噬细胞极化并改变糖尿病肾病中外泌体 miRNA 的表达。
Clin Lab. 2021 Dec 1;67(12). doi: 10.7754/Clin.Lab.2021.210314.
8
CASQ2 alleviates lung cancer by inhibiting M2 tumor-associated macrophage polarization and JAK/STAT pathway.钙网织蛋白 2 通过抑制 M2 肿瘤相关巨噬细胞极化和 JAK/STAT 通路缓解肺癌。
J Biochem Mol Toxicol. 2024 Aug;38(8):e23801. doi: 10.1002/jbt.23801.
9
A hispanolone-derived diterpenoid inhibits M2-Macrophage polarization in vitro via JAK/STAT and attenuates chitin induced inflammation in vivo.一种来源于 Hispanolone 的二萜抑制 M2 巨噬细胞体外极化,通过 JAK/STAT 并减轻体内几丁质诱导的炎症。
Biochem Pharmacol. 2018 Aug;154:373-383. doi: 10.1016/j.bcp.2018.06.002. Epub 2018 Jun 2.
10
Naoqing formula alleviates cerebral ischemia/reperfusion injury induced inflammatory injury by regulating Csf3 mediated JAK/STAT pathway and macrophage polarization.脑清方通过调节Csf3介导的JAK/STAT信号通路和巨噬细胞极化减轻脑缺血/再灌注损伤诱导的炎症损伤。
Phytomedicine. 2025 May;140:156626. doi: 10.1016/j.phymed.2025.156626. Epub 2025 Mar 9.

本文引用的文献

1
S-propargyl-cysteine attenuates temporomandibular joint osteoarthritis by regulating macrophage polarization via Inhibition of JAK/STAT signaling.S-炔丙基半胱氨酸通过抑制JAK/STAT信号通路调节巨噬细胞极化来减轻颞下颌关节骨关节炎。
Mol Med. 2025 Apr 7;31(1):128. doi: 10.1186/s10020-025-01186-6.