Gomori J M, Grossman R I, Goldberg H I, Zimmerman R A, Bilaniuk L T
Radiology. 1985 Oct;157(1):87-93. doi: 10.1148/radiology.157.1.4034983.
Twenty intracranial hematomas between 1 day and over 1 year old were imaged using magnetic resonance at 1.5 T, with T1- and T2-weighted spin-echo pulse sequences. Characteristic intensity patterns were observed in the evolution of the hematomas, which could be staged as acute (less than 1 week old), subacute (greater than 1 week and less than 1 month old), or chronic (greater than 1 month old). Acute hematomas were characterized by central hypointensity on T2-weighted images (WIs). Subacute hematomas had peripheral hyperintensity on T1-WIs and then on T2-WIs. This hyperintensity proceeded to fill in the hematoma in the chronic stage. In subacute and chronic hematomas, there was hypointensity on T2-WIs in the immediately adjacent part of the brain. On T2-WIs of acute and subacute hematomas, the nearby white matter was characterized by hyperintensity, consistent with edema. A different mechanism is proposed for each of the three characteristic intensity patterns. Two of these mechanisms increase in proportion to the square of the magnetic field magnitude.
使用1.5T磁共振成像仪，通过T1加权和T2加权自旋回波脉冲序列，对20例年龄在1天至1岁以上的颅内血肿进行成像。在血肿演变过程中观察到特征性的信号强度模式，可分为急性期（小于1周龄）、亚急性期（大于1周且小于1月龄）或慢性期（大于1月龄）。急性期血肿在T2加权图像（WI）上表现为中央低信号。亚急性期血肿在T1加权图像上随后在T2加权图像上出现周边高信号。这种高信号在慢性期逐渐填充血肿。在亚急性和慢性血肿中，紧邻血肿的脑区在T2加权图像上呈低信号。在急性和亚急性血肿的T2加权图像上，附近白质表现为高信号，与水肿相符。针对这三种特征性信号强度模式分别提出了不同的机制。其中两种机制与磁场强度的平方成正比。
