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本文引用的文献
1
Studies of Slc30a10 Deficiency in Mice Reveal That Intestinal Iron Transporters Dmt1 and Ferroportin Transport Manganese.
Cell Mol Gastroenterol Hepatol. 2025;19(7):101489. doi: 10.1016/j.jcmgh.2025.101489. Epub 2025 Feb 28.
2
Comparative analysis of the functional properties of human and mouse ferroportin.
Am J Physiol Cell Physiol. 2023 May 1;324(5):C1110-C1118. doi: 10.1152/ajpcell.00063.2023. Epub 2023 Mar 20.
3
Regulation of the Metal Transporters ZIP14 and ZnT10 by Manganese Intake in Mice.
Nutrients. 2019 Sep 4;11(9):2099. doi: 10.3390/nu11092099.
4
Mice overexpressing hepcidin suggest ferroportin does not play a major role in Mn homeostasis.
Metallomics. 2019 May 22;11(5):959-967. doi: 10.1039/c8mt00370j.
5
Mutations in SLC39A14 disrupt manganese homeostasis and cause childhood-onset parkinsonism-dystonia.
Nat Commun. 2016 May 27;7:11601. doi: 10.1038/ncomms11601.
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Mn(2+)-mediated homogeneous Fenton-like reaction of Fe(III)-NTA complex for efficient degradation of organic contaminants under neutral conditions.
J Hazard Mater. 2016 Aug 5;313:193-200. doi: 10.1016/j.jhazmat.2016.04.003. Epub 2016 Apr 4.
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SLC39A8 Deficiency: A Disorder of Manganese Transport and Glycosylation.
Am J Hum Genet. 2015 Dec 3;97(6):894-903. doi: 10.1016/j.ajhg.2015.11.003.
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Divalent metal transporter 1 (Dmt1) mediates copper transport in the duodenum of iron-deficient rats and when overexpressed in iron-deprived HEK-293 cells.
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Dystonia with brain manganese accumulation resulting from SLC30A10 mutations: a new treatable disorder.
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