Intestinal Iron Transporters Facilitate Manganese Absorption Under Non-physiological Conditions.
作者信息
Collins James F
机构信息
Food Science & Human Nutrition Department, University of Florida, Gainesville, Florida.
出版信息
Cell Mol Gastroenterol Hepatol. 2025;19(7):101519. doi: 10.1016/j.jcmgh.2025.101519. Epub 2025 May 9.
Abstract
摘要
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本文引用的文献
1
Studies of Slc30a10 Deficiency in Mice Reveal That Intestinal Iron Transporters Dmt1 and Ferroportin Transport Manganese.对小鼠中Slc30a10缺乏的研究表明，肠道铁转运蛋白Dmt1和铁转运蛋白运输锰。
Cell Mol Gastroenterol Hepatol. 2025;19(7):101489. doi: 10.1016/j.jcmgh.2025.101489. Epub 2025 Feb 28.
2
Comparative analysis of the functional properties of human and mouse ferroportin.比较分析人源和鼠源铁蛋白的功能特性。
Am J Physiol Cell Physiol. 2023 May 1;324(5):C1110-C1118. doi: 10.1152/ajpcell.00063.2023. Epub 2023 Mar 20.
3
Regulation of the Metal Transporters ZIP14 and ZnT10 by Manganese Intake in Mice.锰摄入对小鼠金属转运蛋白 ZIP14 和 ZnT10 的调节。
Nutrients. 2019 Sep 4;11(9):2099. doi: 10.3390/nu11092099.
4
Mice overexpressing hepcidin suggest ferroportin does not play a major role in Mn homeostasis.过表达铁调素的小鼠表明铁蛋白转运体在锰稳态中不起主要作用。
Metallomics. 2019 May 22;11(5):959-967. doi: 10.1039/c8mt00370j.
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Mutations in SLC39A14 disrupt manganese homeostasis and cause childhood-onset parkinsonism-dystonia.SLC39A14 基因突变破坏锰离子代谢平衡，导致儿童起病的帕金森病-肌张力障碍。
Nat Commun. 2016 May 27;7:11601. doi: 10.1038/ncomms11601.
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Mn(2+)-mediated homogeneous Fenton-like reaction of Fe(III)-NTA complex for efficient degradation of organic contaminants under neutral conditions.在中性条件下，通过 Mn(2+)介导的 Fe(III)-NTA 配合物均相 Fenton 样反应高效降解有机污染物。
J Hazard Mater. 2016 Aug 5;313:193-200. doi: 10.1016/j.jhazmat.2016.04.003. Epub 2016 Apr 4.
7
SLC39A8 Deficiency: A Disorder of Manganese Transport and Glycosylation.溶质载体家族39成员8缺乏症：一种锰转运和糖基化紊乱疾病。
Am J Hum Genet. 2015 Dec 3;97(6):894-903. doi: 10.1016/j.ajhg.2015.11.003.
8
Intestinal DMT1 is critical for iron absorption in the mouse but is not required for the absorption of copper or manganese.肠道二价金属离子转运体1对小鼠铁吸收至关重要，但对铜或锰的吸收并非必需。
Am J Physiol Gastrointest Liver Physiol. 2015 Oct 15;309(8):G635-47. doi: 10.1152/ajpgi.00160.2015. Epub 2015 Aug 20.
9
Divalent metal transporter 1 (Dmt1) mediates copper transport in the duodenum of iron-deficient rats and when overexpressed in iron-deprived HEK-293 cells.二价金属转运蛋白 1（Dmt1）在缺铁大鼠的十二指肠中介导铜的转运，并且在缺铁的 HEK-293 细胞中过表达。
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Dystonia with brain manganese accumulation resulting from SLC30A10 mutations: a new treatable disorder.由于 SLC30A10 基因突变导致的伴脑锰沉积的肌张力障碍：一种新的可治疗疾病。
Mov Disord. 2012 Sep 1;27(10):1317-22. doi: 10.1002/mds.25138. Epub 2012 Aug 23.
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