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外周化学感受器在对烟雾诱导的呼吸暂停与气管阻塞反应中的作用。

Role of peripheral chemoreceptors in response to smoke-induced apnea vs tracheal occlusion.

作者信息

O'Dell S L, Peterson D F

出版信息

Experientia. 1985 Sep 15;41(9):1130-1. doi: 10.1007/BF01951692.

DOI:10.1007/BF01951692
PMID:4043319
Abstract

Reflex autonomic changes which occur after cigarette smoke enters the upper airways are partially due to peripheral chemoreceptor stimulation. Chemoreceptor denervation attenuates but does not abolish smoke induced bradycardia. Denervation nearly abolishes bradycardia induced by tracheal occlusion. Hypertension accompanies smoke induced apnea but does not occur during tracheal occlusion.

摘要

香烟烟雾进入上呼吸道后发生的反射性自主神经变化部分归因于外周化学感受器刺激。化学感受器去神经支配可减弱但不能消除烟雾诱导的心动过缓。去神经支配几乎可消除气管阻塞诱导的心动过缓。高血压伴随烟雾诱导的呼吸暂停出现,但在气管阻塞期间不发生。

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