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本文引用的文献
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Genome-Wide DNA Methylation Profiling Reveals Low Methylation Variability in Moyamoya Disease.
Transl Stroke Res. 2024 Oct 2. doi: 10.1007/s12975-024-01299-w.
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Adult Moyamoya Disease and Syndrome: Current Perspectives and Future Directions: A Scientific Statement From the American Heart Association/American Stroke Association.
Stroke. 2023 Oct;54(10):e465-e479. doi: 10.1161/STR.0000000000000443. Epub 2023 Aug 23.
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RNF213 loss-of-function promotes pathological angiogenesis in moyamoya disease via the Hippo pathway.
Brain. 2023 Nov 2;146(11):4674-4689. doi: 10.1093/brain/awad225.
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Vulnerability to shear stress caused by altered peri-endothelial matrix is a key feature of Moyamoya disease.
Sci Rep. 2021 Jan 15;11(1):1552. doi: 10.1038/s41598-021-81282-9.
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Regulatory T Cells in Angiogenesis.
J Immunol. 2020 Nov 15;205(10):2557-2565. doi: 10.4049/jimmunol.2000574.
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Brain transforms natural killer cells that exacerbate brain edema after intracerebral hemorrhage.
J Exp Med. 2020 Dec 7;217(12). doi: 10.1084/jem.20200213.
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Aberrant Promoter Hypomethylation of Sortilin 1: A Moyamoya Disease Biomarker.
J Stroke. 2018 Sep;20(3):350-361. doi: 10.5853/jos.2018.00962. Epub 2018 Sep 30.
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Association of increased Treg and Th17 with pathogenesis of moyamoya disease.
Sci Rep. 2017 Jun 8;7(1):3071. doi: 10.1038/s41598-017-03278-8.
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CLEVER-1 mediates lymphocyte transmigration through vascular and lymphatic endothelium.
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FEEL-1, a novel scavenger receptor with in vitro bacteria-binding and angiogenesis-modulating activities.
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