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[实验性静脉血栓形成:光凝前后视网膜前氧分压]

[Experimental venous thrombosis: preretinal PO2 before and after photocoagulation].

作者信息

Pournaras C J, Ilic J, Gilodi N, Tsacopoulos M, Leuenberger M P

出版信息

Klin Monbl Augenheilkd. 1985 Jun;186(6):500-1. doi: 10.1055/s-2008-1050970.

Abstract

Experimental occlusion of a retinal vein leads to retinopathy in the affected drainage area. In some cases the formation of ischemic regions, where the retina is completely destroyed, is typical for the development of this retinopathy. Laser photocoagulation restores the physiological preretinal oxygen pressure in these hypoxic regions. It is possible that the rise in O2 pressure which has been observed is caused by destruction of pigment epithelium and the photoreceptor layer, as oxygen diffuses freely from the choroid to the inner retina without being consumed by the outer layers.

摘要

实验性视网膜静脉阻塞会导致受影响引流区域出现视网膜病变。在某些情况下,视网膜完全受损的缺血区域的形成是这种视网膜病变发展的典型特征。激光光凝可恢复这些缺氧区域视网膜前的生理氧分压。观察到的氧分压升高可能是由于色素上皮和光感受器层被破坏所致,因为氧气可从脉络膜自由扩散至视网膜内层,而不会被外层消耗。

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