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[两例巨大梭形动脉瘤引发缺血性发作]

[Two cases of giant fusiform aneurysm presenting ischemic attacks].

作者信息

Kobayashi N, Nakagawa Y, Sakuragi M, Tashiro K, Abe H, Tsuru M, Nomura M, Ueno K

出版信息

No Shinkei Geka. 1985 Aug;13(8):919-25.

PMID:4058669
Abstract

It is said that major clinical sign of giant fusiform aneurysm is so-called mass sign compressing surrounding tissues and ischemic attack caused by the aneurysm is very rare. The authors experienced such uncommon two patients with partially thrombosed fusiform aneurysm who developed ischemic attack. Case 1: A 68-year-old man was admitted to our hospital in semicomatous state with ocular bobbing and decorticate posture of sudden onset. Enhanced CT scans taken at admission showed a presence of homogenously enhanced mass continuing to bilateral posterior cerebral arteries, which compressed the pons from right ventro-lateral side (Fig. 1 a,b). Plain CT scan taken 10 days after admission demonstrated extensive low density area in the pons (Fig. 1c). Basilar artery and P1 segment of the right posterior cerebral artery are ectatic, tortuous and elongated (Fig. 2a). The ectatic basilar artery was displaced upward and posteriorly (Fig. 2b). He developed locked-in syndrome and died of pneumonia six months after admission. At autopsy, large aneurysm measuring 3.5 X 2.0 X 1.9 cm was verified in the basilar artery (Fig. 3a). Cross section of the basilar artery aneurysm showed that laminated thrombosis nearly occupied the lumen with partially unobstructed portion (Fig. 3b). Also, horizontal section of the pons demonstrated a presence of old infarction located in the perforating arteries territory (Fig. 3c). Case 2: A 42-year-old man was initially hospitalized in 1978 because of multiple intracranial meningiomas and subtotal removal of meningiomas located in the tuberculum sellae and the foramen magnum was performed.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

据说,巨大梭形动脉瘤的主要临床体征是所谓的肿块征,即压迫周围组织,而由动脉瘤引起的缺血性发作非常罕见。作者遇到了两名不常见的部分血栓形成的梭形动脉瘤患者,他们出现了缺血性发作。病例1:一名68岁男性,以突发的半昏迷状态、眼球摆动和去皮质姿势入院。入院时的增强CT扫描显示,有一个均匀强化的肿块,延续至双侧大脑后动脉,从右侧腹外侧压迫脑桥(图1a、b)。入院10天后的平扫CT扫描显示脑桥有广泛的低密度区(图1c)。基底动脉和右侧大脑后动脉的P1段扩张、迂曲且延长(图2a)。扩张的基底动脉向上和向后移位(图2b)。他发展为闭锁综合征,并在入院6个月后死于肺炎。尸检时,在基底动脉中证实了一个大小为3.5×2.0×1.9cm的大动脉瘤(图3a)。基底动脉动脉瘤的横截面显示,分层血栓几乎占据了管腔,部分区域未阻塞(图3b)。此外,脑桥的水平切片显示,在穿通动脉区域有陈旧性梗死(图3c)。病例2:一名42岁男性,1978年因多发性颅内脑膜瘤首次住院,并对位于鞍结节和枕骨大孔的脑膜瘤进行了次全切除。(摘要截断于250字)

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