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区分轻度、重度和非典型过氧化物酶体生物发生障碍中PEX2和PEX16基因变异的严重程度。

Distinguishing PEX2 and PEX16 gene variant severity for mild, severe and atypical peroxisome biogenesis disorders.

作者信息

Gomez Vanessa A, Kanca Oguz, Jangam Sharayu V, Srivastav Saurabh, Andrews Jonathan C, Wangler Michael F

机构信息

Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.

Jan and Dan Duncan Neurological Research Institute, Texas Children's Hospital, Houston, TX 77030, USA.

出版信息

Dis Model Mech. 2025 Jul 1;18(7). doi: 10.1242/dmm.052258. Epub 2025 Jul 28.

DOI:10.1242/dmm.052258
PMID:40621817
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12352285/
Abstract

Peroxisomal biogenesis disorders (PBD) are autosomal recessive diseases caused by mutations in specific PEX genes that impair peroxisome formation, leading to multi-systemic failure. Symptoms vary, even in patients with variants in the same PEX gene. Our goal is to select PEX mutations and use Drosophila to model a severity spectrum based on genotype-phenotype correlations. Utilizing KozakGAL4 (KZ) cassettes, we replaced the coding sequence of Pex with a GAL4 driver, ideal for making 'humanized' flies in which human PEX can replace the fly loss. We generated Pex2KZ and Pex16KZ lines and assessed them in various behavior assays, confirming their severe phenotypes. We performed rescue with human reference, variant PEX2 and PEX16 alleles, and phenotypic rescue was observed when human PEX2Ref or PEX16Ref were expressed in Pex2KZ or Pex16KZ flies, respectively. We identified a severity spectrum for PEX2 and PEX16 alleles, with some missense mutations exhibiting severity comparable to truncations. Alleles linked to mild PBD showed partial rescue, while variants associated with atypical ataxia could fully rescue. Drosophila humanization is an effective method to study the range of severity of PBD.

摘要

过氧化物酶体生物发生障碍(PBD)是由特定PEX基因突变引起的常染色体隐性疾病,这些突变会损害过氧化物酶体的形成,导致多系统功能衰竭。症状各不相同,即使是同一PEX基因存在变异的患者也是如此。我们的目标是选择PEX突变,并利用果蝇根据基因型-表型相关性建立严重程度谱模型。利用KozakGAL4(KZ)盒,我们用GAL4驱动子取代了Pex的编码序列,这对于制造“人源化”果蝇非常理想,在这种果蝇中人类PEX可以替代果蝇缺失的部分。我们生成了Pex2KZ和Pex16KZ品系,并在各种行为试验中对它们进行评估,证实了它们的严重表型。我们用人源参考、变异的PEX2和PEX16等位基因进行拯救实验,当人源PEX2Ref或PEX16Ref分别在Pex2KZ或Pex16KZ果蝇中表达时,观察到了表型拯救。我们确定了PEX2和PEX16等位基因的严重程度谱,一些错义突变表现出与截短突变相当的严重程度。与轻度PBD相关的等位基因表现出部分拯救,而与非典型共济失调相关的变异则可以完全拯救。果蝇人源化是研究PBD严重程度范围的一种有效方法。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/aba8d22fe379/dmm-18-052258-g6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/4afac99cb7ce/dmm-18-052258-g1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/31d143ac3e39/dmm-18-052258-g2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/b40eac4ef157/dmm-18-052258-g3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/7374dcd22386/dmm-18-052258-g4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/9fc052492210/dmm-18-052258-g5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/aba8d22fe379/dmm-18-052258-g6.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/4afac99cb7ce/dmm-18-052258-g1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/31d143ac3e39/dmm-18-052258-g2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/b40eac4ef157/dmm-18-052258-g3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/7374dcd22386/dmm-18-052258-g4.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/9fc052492210/dmm-18-052258-g5.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d4dd/12352285/aba8d22fe379/dmm-18-052258-g6.jpg

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