Kokova N I
Kosm Biol Aviakosm Med. 1985 Sep-Oct;19(5):56-60.
Hemodynamic parameters in the subjects with various resistance to +Gz acceleration were investigated. Twelve test subjects were rotated 41 times in a 7.25 m-arm centrifuge. The acceleration value was increased slowly. During centrifugation ECG, systolic pressure in the ear lobe, stroke volume and cardiac output were measured. In the resistant subjects stroke volume remained stable and cardiac output was close to or higher than the initial level (due to tachycardia). In the nonresistant subjects both parameters decreased although their heart rate was significantly higher than in the resistant subjects. Various visual disorders developed when cardiac output decreased by 50-70% as compared to the initial level. It is concluded that in the nonresistant people the compensatory mechanisms responsible for the stability of cardiac output include primarily HR increase, whereas in the resistant people they involve a high level of venous return and stroke volume.
对不同 +Gz 加速度耐力受试者的血流动力学参数进行了研究。12 名受试对象在臂长 7.25 米的离心机中旋转了 41 次。加速度值缓慢增加。在离心过程中，测量了心电图、耳垂收缩压、每搏输出量和心输出量。在耐力较强的受试者中，每搏输出量保持稳定，心输出量接近或高于初始水平（由于心动过速）。在耐力较弱的受试者中，尽管他们的心率明显高于耐力较强的受试者，但这两个参数均下降。当心输出量比初始水平降低 50 - 70% 时，会出现各种视觉障碍。得出的结论是，在耐力较弱的人群中，负责心输出量稳定的代偿机制主要包括心率增加，而在耐力较强的人群中，这些机制涉及高水平的静脉回流和每搏输出量。
