Katafuchi Y, Nishimi T, Yamaguchi Y, Matsuishi T, Kimura Y, Otaki E, Yamashita Y
Brain Dev. 1985;7(5):516-9. doi: 10.1016/s0387-7604(85)80121-2.
A 3-year-old boy had persistent cortical blindness following acute carbon monoxide poisoning. He was believed to have suffered anoxic brain damage due to incomplete combustion of the briquette-type solid fuel. Computed tomographic (CT) scan of the brain and visual evoked potentials (VEP) in the early stage were normal. However, on the 20th hospital day CT scan showed leukomalacia and VEP showed an absence of N1-, and P1-waves which was well correlated with the clinical feature at that time.
一名3岁男孩在急性一氧化碳中毒后持续存在皮质盲。据信他因煤球型固体燃料不完全燃烧而遭受缺氧性脑损伤。早期脑部计算机断层扫描（CT）和视觉诱发电位（VEP）均正常。然而，在住院第20天时，CT扫描显示脑白质软化，VEP显示N1波和P1波缺失，这与当时的临床特征密切相关。
