Mafi Pouya, Konda Nagarjun N, Venus Matthew
Plastic Surgery, University Hospitals Coventry and Warwickshire NHS Trust, Coventry, GBR.
Cureus. 2025 Aug 23;17(8):e90819. doi: 10.7759/cureus.90819. eCollection 2025 Aug.
Paediatric trigger finger (PTF) is a rare condition, far less common than paediatric trigger thumb, and often associated with anatomical anomalies or systemic disease. Unlike trigger thumb, which may resolve spontaneously, PTF usually requires surgical treatment. Trauma is an uncommon cause, and closed intratendinous rupture of the flexor digitorum profundus (FDP) has not previously been reported in a child following direct injury. We present the case of a healthy 12-year-old boy who developed triggering of the right middle finger eight weeks after blunt trauma during rugby. Examination revealed a palpable nodule at the A2 pulley and paradoxical distal interphalangeal (DIP) joint extension during fist-making (lumbrical plus phenomenon). Ultrasound and MRI demonstrated intact FDP and flexor digitorum superficialis (FDS) tendons without an obvious lesion. Ongoing symptoms led to surgical exploration, which revealed a partial intratendinous FDP rupture with a scar nodule beneath an intact A2 pulley. Management included partial A2 pulley release, nodule excision, FDP repair with 5/0 polypropylene suture, and lumbrical release. Early active mobilisation began within days postoperatively. At one year, the patient had a full, pain-free range of motion, normal grip strength, and no recurrence. This is the first reported paediatric case of PTF caused by a closed, trauma-induced partial FDP rupture. The intratendinous tear produced a scar mass that impinged beneath the A2 pulley, mimicking more typical A1 pulley pathology. The lumbrical plus sign, rarely described in children and usually linked to complete ruptures, here resulted from a partial tendon injury. Standard imaging did not identify the lesion, highlighting the limitations of ultrasound and MRI in subtle intratendinous injuries. Careful clinical examination, particularly eliciting paradoxical extension, was critical to diagnosis. Literature review indicates most PTF cases are idiopathic or related to systemic disease, with only ~5% linked to trauma. Surgical release yields higher resolution rates than conservative management (87% vs. 58%). Given the mechanical nature and delayed presentation of this lesion, surgery was both diagnostic and therapeutic. A comprehensive approach, pulley release, tendon repair, and lumbrical release, produced an excellent outcome. PTF after blunt trauma can arise from closed intratendinous FDP rupture, even when imaging is unremarkable. The lumbrical plus sign is a valuable but under-recognised diagnostic clue. Early surgical exploration should be considered when suspicion remains high, as timely intervention can restore full function and prevent long-term disability.
小儿扳机指(PTF)是一种罕见病症,远比小儿扳机拇少见,且常与解剖学异常或全身性疾病相关。与可能自行缓解的扳机拇不同,PTF通常需要手术治疗。创伤是一种不常见的病因,此前尚未有儿童因直接损伤导致指深屈肌(FDP)闭合性肌腱内断裂的报道。我们报告一例健康的12岁男孩,在橄榄球运动中钝性外伤八周后出现右手中指扳机现象。检查发现A2滑车处可触及结节,握拳时出现矛盾性远侧指间关节(DIP)伸展(蚓状肌加现象)。超声和磁共振成像(MRI)显示FDP和指浅屈肌(FDS)肌腱完整,无明显病变。持续的症状导致进行手术探查,结果显示FDP肌腱内部分断裂,完整的A2滑车下方有瘢痕结节。治疗包括部分A2滑车松解、结节切除、用5-0聚丙烯缝线修复FDP以及蚓状肌松解。术后数天内即开始早期主动活动。一年后,患者活动范围完全正常、无痛,握力正常,且无复发。这是首例报道的因闭合性创伤性FDP部分断裂导致的小儿PTF病例。肌腱内撕裂产生的瘢痕团块在A2滑车下方受到挤压,类似于更典型的A1滑车病变。蚓状肌加征在儿童中很少被描述,通常与完全断裂有关,在此病例中是由部分肌腱损伤所致。标准影像学检查未发现病变,凸显了超声和MRI在细微肌腱内损伤诊断中的局限性。仔细的临床检查,尤其是引出矛盾性伸展,对诊断至关重要。文献综述表明,大多数PTF病例为特发性或与全身性疾病相关,仅有约5%与创伤有关。手术松解的治愈率高于保守治疗(87%对58%)。鉴于该病变的机械性质和延迟表现,手术兼具诊断和治疗作用。综合治疗方法,包括滑车松解、肌腱修复和蚓状肌松解,取得了极佳的效果。钝性外伤后的PTF可能由FDP闭合性肌腱内断裂引起,即使影像学检查无明显异常。蚓状肌加征是一个有价值但未得到充分认识的诊断线索。当高度怀疑时应考虑早期手术探查,因为及时干预可恢复全部功能并防止长期残疾。