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Retinal changes induced by treatment with vincristine and vinblastine.

作者信息

Hansson H A

出版信息

Doc Ophthalmol. 1972 Apr 15;31(1):65-88. doi: 10.1007/BF00167362.

DOI:10.1007/BF00167362
PMID:4111990
Abstract
摘要

相似文献

1
Retinal changes induced by treatment with vincristine and vinblastine.
Doc Ophthalmol. 1972 Apr 15;31(1):65-88. doi: 10.1007/BF00167362.
2
Ultrastructural changes induced in the rat retina by treatment with colchicine.秋水仙碱处理诱导大鼠视网膜的超微结构变化。
Doc Ophthalmol. 1972 Apr 15;31(1):29-63. doi: 10.1007/BF00167361.
3
Protein synthesis in ganglion cells of the rabbit retina after intravitreous injection of vinblastine.
Invest Ophthalmol. 1973 Jun;12(6):467-9.
4
Ultrastructural changes of secreting rat-incisor ameloblasts following administration of vincristine and vinblastine.
J Dent Res. 1982 Dec;Spec No:1472-8.
5
[Ultrastructure changes in ameloblasts of rats administered with vincristine and vinblastine (author's transl)].
Shikwa Gakuho. 1982 Mar;82(3):347-83.
6
Axoplasmic transport.
Comp Biochem Physiol A Comp Physiol. 1975 Aug 1;51(4):701-4. doi: 10.1016/0300-9629(75)90041-9.
7
Vinblastine-induced blockage of orthograde and retrograde axonal transport of protein in retinal ganglion cells.长春碱诱导的视网膜神经节细胞中蛋白质顺向和逆向轴突运输的阻断。
Exp Neurol. 1974 Nov;45(2):288-97. doi: 10.1016/0014-4886(74)90119-8.
8
Effects of two mitosis inhibitors (colchicine and vinblastine) on the distribution and axonal transport of noradrenaline storage particles, studied by fluorescence and electron microscopy.通过荧光显微镜和电子显微镜研究两种有丝分裂抑制剂(秋水仙碱和长春碱)对去甲肾上腺素储存颗粒的分布及轴突运输的影响。
Z Zellforsch Mikrosk Anat. 1971;119(4):460-82. doi: 10.1007/BF00455243.
9
Effects of vinblastine and colchicine on monoamine containing neurons of the rat, with special regard to the axoplasmic transport of amine granules.长春碱和秋水仙碱对大鼠含单胺神经元的作用,特别关注胺颗粒的轴浆运输。
Acta Neuropathol. 1971;5:Suppl 5:226-37. doi: 10.1007/978-3-642-47449-1_28.
10
Cytochalasin B and the intra-axonal movement of noradrenaline storage vesicles.细胞松弛素B与去甲肾上腺素储存囊泡的轴突内移动
Brain Res. 1973 Jan 30;49(2):417-21. doi: 10.1016/0006-8993(73)90433-2.

引用本文的文献

1
Retinal and optic nerve atrophy induced by intravitreous vincristine in the primate.灵长类动物玻璃体内注射长春新碱所致的视网膜和视神经萎缩
Trans Am Ophthalmol Soc. 1975;73:389-416.
2
[Changes in the outer plexiform layer of the rabbit retina after long-term application of vincristine].[长期应用长春新碱后兔视网膜外网状层的变化]
Albrecht Von Graefes Arch Klin Exp Ophthalmol. 1979 Jul 2;211(1):23-33. doi: 10.1007/BF00414651.

本文引用的文献

1
The renewal of photoreceptor cell outer segments.光感受器细胞外节的更新。
J Cell Biol. 1967 Apr;33(1):61-72. doi: 10.1083/jcb.33.1.61.
2
Microtubular crystals in mammalian cells.哺乳动物细胞中的微管晶体。
J Cell Biol. 1969 Jan;40(1):95-107. doi: 10.1083/jcb.40.1.95.
3
The effect of colchicine on the axonal transport of protein in the optic nerve and tract of the rabbit.秋水仙碱对家兔视神经和视束中蛋白质轴突运输的影响。
Brain Res. 1969 May;13(3):617-9. doi: 10.1016/0006-8993(69)90274-1.
4
Neurofibrillary degeneration induced by vincristine therapy.长春新碱治疗引起的神经原纤维变性。
Arch Neurol. 1969 Feb;20(2):199-206. doi: 10.1001/archneur.1969.00480080099012.
5
Ultrastructural studies on tissue culture cells treated with vincristine (NSC-67574).用长春新碱(NSC - 67574)处理的组织培养细胞的超微结构研究。
Cancer Chemother Rep. 1968 Jun;52(4):509-17.
6
Neuronal changes induced by intrathecal vincristine sulfate.鞘内注射硫酸长春新碱引起的神经元变化。
J Neuropathol Exp Neurol. 1968 Oct;27(4):645-58.
7
The renewal of protein in retinal rods and cones.视网膜视杆细胞和视锥细胞中蛋白质的更新。
J Cell Biol. 1968 Oct;39(1):169-84. doi: 10.1083/jcb.39.1.169.
8
Hypertrophy of granular endoplasmic reticulum and annulate lamellae in Earle's L cells exposed to vinblastine sulfate.暴露于硫酸长春碱的厄尔L细胞中粗面内质网和环孔板的肥大。
J Cell Biol. 1968 Oct;39(1):211-6. doi: 10.1083/jcb.39.1.211.
9
Effects of colchicine and Vinca alkaloids on human platelets. I. Influence on platelet microtubules and contractile function.秋水仙碱和长春花生物碱对人血小板的作用。I. 对血小板微管和收缩功能的影响。
Am J Pathol. 1968 Aug;53(2):281-91.
10
Ameboid movement induced in cultured macrophages by colchicine or vinblastine.秋水仙碱或长春花碱在培养的巨噬细胞中诱导的阿米巴样运动。
Exp Cell Res. 1971 Feb;64(2):419-29. doi: 10.1016/0014-4827(71)90096-6.