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The dysequilibrium syndrome in cerebral palsy. Clinical aspects and treatment.

作者信息

Hagberg B, Sanner G, Steen M

出版信息

Acta Paediatr Scand Suppl. 1972;226:1-63.

PMID:4115893
Abstract
摘要

相似文献

1
The dysequilibrium syndrome in cerebral palsy. Clinical aspects and treatment.脑瘫中的平衡失调综合征。临床症状与治疗。
Acta Paediatr Scand Suppl. 1972;226:1-63.
2
[Early diagnosis and therapy of cerebral movement disorders in childhood. C. Reflexogenous locomotion--reflex creeping and reflex turning. C2. Its use in 207 risk children. Analysis of the final results].[儿童期脑性运动障碍的早期诊断与治疗。C. 反射性运动——反射性爬行与反射性翻身。C2. 其在207名高危儿童中的应用。最终结果分析]
Z Orthop Ihre Grenzgeb. 1973 Jun;111(3):292-309.
3
[School problems in minimal cerebral palsy].
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4
A pediatric overview of the spectrum of developmental disabilities.发育障碍谱系的儿科概述。
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7
[Early diagnosis and therapy of cerebral movement disorders in childhood. B. The developmental diagnosis].[儿童期脑性运动障碍的早期诊断与治疗。B. 发育诊断]
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Is paediatric assessment of motor development of very preterm and low-birthweight children appropriate?对极早产儿和低体重儿进行运动发育的儿科评估是否合适？
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[Diagnosis of development disorders in infants and small children].
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两个患有四足行走、智力发育迟缓、无语言能力和婴儿期肌张力减退（乌纳·坦综合征II型）的家庭；关于人类双足行走进化出现的一种新理论。
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Novel VLDLR microdeletion identified in two Turkish siblings with pachygyria and pontocerebellar atrophy.在两名患有巨脑回和桥脑小脑萎缩的土耳其兄弟中发现了新型 VLDLR 微缺失。
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Mutations in VLDLR as a cause for autosomal recessive cerebellar ataxia with mental retardation (dysequilibrium syndrome).极低密度脂蛋白受体（VLDLR）突变是常染色体隐性遗传性小脑共济失调伴智力发育迟缓（失衡综合征）的病因。
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Reply to Herz et al. and Humphrey et al.: Genetic heterogeneity of cerebellar hypoplasia with quadrupedal locomotion.对赫茨等人以及汉弗莱等人的回复：伴有四足运动的小脑发育不全的遗传异质性。
Proc Natl Acad Sci U S A. 2008 Jun 10;105(23):E32-3. doi: 10.1073/pnas.0804078105.
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Mutations in the very low-density lipoprotein receptor VLDLR cause cerebellar hypoplasia and quadrupedal locomotion in humans.极低密度脂蛋白受体（VLDLR）的突变会导致人类小脑发育不全和四肢行走。
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